137739 20240321220314.0 10.1161/CIRCRESAHA.116.301135 doi pmid:25593277 pmid 0009-7330 ISSN 1524-4571 ISSN altmetric:3069715 altmetric DZNE-2020-04061 English 610 Zimmer, Sebastian P:(DE-HGF)0 0 Danger signaling in atherosclerosis. New York, NY 2015 Assoc. print Crossref Ovid Technologies (Wolters Kluwer Health) 2015-01-16 article DRIVER Output Types/Journal article DataCite Journal Article journal journal PUB:(DE-HGF)16 1591371143_14981 PUB:(DE-HGF) Review Article ARTICLE BibTeX JOURNAL_ARTICLE ORCID Journal Article 0 EndNote All aspects of the pathogenesis of atherosclerosis are critically influenced by the inflammatory response in vascular plaques. Research in the field of innate immunity from the past 2 decades has uncovered many novel mechanisms elucidating how immune cells sense microbes, tissue damage, and metabolic derangements. Here, we summarize which triggers of innate immunity appear during atherogenesis and by which pathways they can contribute to inflammation in atherosclerotic plaques. The increased understanding gained from studies assessing how immune activation is associated with the pathogenesis of atherosclerosis has provided many novel targets for potential therapeutic intervention. Excitingly, the concept that inflammation may be the core of cardiovascular disease is currently being clinically evaluated and will probably encourage further studies in this area. 342 - Disease Mechanisms and Model Systems (POF3-342) G:(DE-HGF)POF3-342 POF3-342 POF III 0 Dataset connected to CrossRef, PubMed, Animals MeSH Atherosclerosis: etiology MeSH Atherosclerosis: immunology MeSH Atherosclerosis: microbiology MeSH Humans MeSH Immunity, Innate: immunology MeSH Signal Transduction: immunology MeSH Grebe, Alena P:(DE-HGF)0 1 Latz, Eicke P:(DE-2719)2000062 2 Last author dzne 10.1161/circresaha.116.301135 : Ovid Technologies (Wolters Kluwer Health), 2015-01-16 2 323-340 journal-article Crossref Circulation Research 116 2015 0009-7330 10.1161/CIRCRESAHA.116.301135 Vol. 116, no. 2, p. 323 - 340 PERI:(DE-600)1467838-X 2 116:2<323 - 340 323-340 Circulation research 116 2015 0009-7330 oai:pub.dzne.de:137739 VDB Deutsches Zentrum für Neurodegenerative Erkrankungen I:(DE-588)1065079516 DZNE 2 P:(DE-2719)2000062 DE-HGF Forschungsbereich Gesundheit Erkrankungen des Nervensystems G:(DE-HGF)POF3-340 G:(DE-HGF)POF3-342 G:(DE-HGF)POF3-300 Disease Mechanisms and Model Systems 0 2015 Allianz-Lizenz StatID:(DE-HGF)0410 StatID Nationallizenz StatID:(DE-HGF)0420 StatID DBCoverage StatID:(DE-HGF)0200 StatID SCOPUS DBCoverage StatID:(DE-HGF)0300 StatID Medline DBCoverage StatID:(DE-HGF)0310 StatID NCBI Molecular Biology Database JCR StatID:(DE-HGF)0100 StatID CIRC RES : 2017 DBCoverage StatID:(DE-HGF)0199 StatID Clarivate Analytics Master Journal List WoS StatID:(DE-HGF)0110 StatID Science Citation Index DBCoverage StatID:(DE-HGF)0150 StatID Web of Science Core Collection WoS StatID:(DE-HGF)0111 StatID Science Citation Index Expanded DBCoverage StatID:(DE-HGF)1030 StatID Current Contents - Life Sciences DBCoverage StatID:(DE-HGF)1050 StatID BIOSIS Previews IF >= 15 StatID:(DE-HGF)9915 StatID CIRC RES : 2017 I:(DE-2719)1013024 AG Latz Innate Immunity in Neurodegeneration 0 journal VDB 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