% IMPORTANT: The following is UTF-8 encoded. This means that in the presence
% of non-ASCII characters, it will not work with BibTeX 0.99 or older.
% Instead, you should use an up-to-date BibTeX implementation like “bibtex8” or
% “biber”.
@ARTICLE{Freire:284093,
author = {Freire, R. H. and Fernandes, L. R. and Silva, R. B. and
Coelho, B. S. L. and de Araújo, L. P. T. and Secchim
Ribeiro, Lucas and Andrade, J. M. O. and Lima, P. M. A. and
Araújo, R. S. and Santos, S. H. S. and Coimbra, C. C. and
Cardoso, V. N. and Alvarez-Leite, J. I.},
title = {{W}heat gluten intake increases weight gain and adiposity
associated with reduced thermogenesis and energy expenditure
in an animal model of obesity},
journal = {International journal of obesity},
volume = {40},
number = {3},
issn = {0307-0565},
address = {Avenel, NJ},
publisher = {Nature Publ. Group},
reportid = {DZNE-2026-00101},
pages = {479 - 486},
year = {2016},
note = {ISSN 1476-5497 not unique: **2 hits**.},
abstract = {The association between gluten and body weight is
inconsistent. Previously, we showed that a gluten-free diet
reduces weight gain without changing food intake in mice fed
high-fat diets. In the present study, we investigated the
effects of gluten intake on fat metabolism, thermogenesis
and energy expenditure in mice fed a standard or high-fat
diet.Mice were fed four different experimental diets during
8 weeks: a control-standard diet (CD), a CD added with
$4.5\%$ of wheat gluten (CD-G), a high-fat diet (HFD) and a
HFD added with $4.5\%$ of wheat gluten (HFD-G). After 8
weeks, the mice received (99m)Tc-radiolabeled gluten orally
to study gluten absorption and biodistribution or they
underwent indirect calorimetry. After killing, subcutaneous
and brown adipose tissues (SAT and BAT) were collected to
assess thermogenesis-related protein expression. Lipid
metabolism was studied in adipocyte cultures from the four
groups.Despite having had the same energy intake, CD-G and
HFD-G mice exhibited increased body weight and fat deposits
compared with their respective controls. (99m)Tc-GLU or its
peptides were detected in the blood, liver and visceral
adipose tissue, suggesting that gluten can even reach
extraintestinal organs. Uncoupling protein-1 expression was
reduced in the BAT of HFD-G and in the SAT of CD-G and HFD-G
mice. Indirect calorimetry showed lower oxygen volume
consumption in CD-G and HFD-G groups compared with their
controls. In HFD mice, daily energy expenditure was reduced
with gluten intake. Gluten also reduced adiponectin,
peroxisome proliferator-activated receptor (PPAR)-α and
PPARγ and hormone-sensitive lipase in cultures of isolated
adipocytes from HFD mice, whereas in the CD-G group, gluten
intake increased interleukin-6 expression and tended to
increase that of tumor necrosis factor.Wheat gluten promotes
weight gain in animals on both HFD and CD, partly by
reducing the thermogenic capacity of adipose tissues.},
keywords = {Adipogenesis / Adiposity / Animals / Disease Models, Animal
/ Energy Intake / Energy Metabolism: physiology / Feeding
Behavior / Gene Expression Regulation / Glutens / Lipid
Metabolism / Male / Mice / Mice, Inbred C57BL / Obesity:
metabolism / Thermogenesis / Weight Gain: physiology /
Glutens (NLM Chemicals)},
ddc = {610},
pnm = {899 - ohne Topic (POF4-899)},
pid = {G:(DE-HGF)POF4-899},
typ = {PUB:(DE-HGF)16},
doi = {10.1038/ijo.2015.204},
url = {https://pub.dzne.de/record/284093},
}