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@ARTICLE{Rmer:135987,
author = {Römer, Benedikt and Krebs, Julia and Overall, Rupert W and
Fabel, Klaus and Babu, Harish and Overstreet-Wadiche, Linda
and Brandt, Moritz D and Williams, Robert W and Jessberger,
Sebastian and Kempermann, Gerd},
title = {{A}dult hippocampal neurogenesis and plasticity in the
infrapyramidal bundle of the mossy fiber projection: {I}.
{C}o-regulation by activity.},
journal = {Frontiers in neuroscience},
volume = {5},
issn = {1662-453X},
address = {Lausanne},
publisher = {Frontiers Research Foundation},
reportid = {DZNE-2020-02309},
pages = {107},
year = {2011},
abstract = {BESIDES THE MASSIVE PLASTICITY AT THE LEVEL OF SYNAPSES, WE
FIND IN THE HIPPOCAMPUS OF ADULT MICE AND RATS TWO SYSTEMS
WITH VERY STRONG MACROSCOPIC STRUCTURAL PLASTICITY: adult
neurogenesis, that is the lifelong generation of new granule
cells, and dynamic changes in the mossy fibers linking the
dentate gyrus to area CA3. In particular the anatomy of the
infrapyramidal mossy fiber tract (IMF) changes in response
to a variety of extrinsic and intrinsic stimuli. Because
mossy fibers are the axons of granule cells, the question
arises whether these two types of plasticity are linked.
Using immunohistochemistry for markers associated with
axonal growth and pro-opiomelanocortin (POMC)-GFP mice to
visualize the post-mitotic maturation phase of adult
hippocampal neurogenesis, we found that newly generated
mossy fibers preferentially but not exclusively contribute
to the IMF. The neurogenic stimulus of an enriched
environment increased the volume of the IMF. In addition,
the IMF grew with a time course consistent with axonal
outgrowth from the newborn neurons after the induction of
neurogenic seizures using kainate. These results indicate
that two aspects of plasticity in the adult hippocampus,
mossy fiber size and neurogenesis, are related and may share
underlying mechanisms. In a second part of this study,
published separately (Krebs et al., 2011) we have addressed
the question of whether there is a shared genetics
underlying both traits.},
cin = {Dresden Pre 2020 / AG Kempermann 1},
ddc = {610},
cid = {I:(DE-2719)6000013 / I:(DE-2719)1710001},
pnm = {342 - Disease Mechanisms and Model Systems (POF3-342)},
pid = {G:(DE-HGF)POF3-342},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:21991243},
pmc = {pmc:PMC3180604},
doi = {10.3389/fnins.2011.00107},
url = {https://pub.dzne.de/record/135987},
}