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@ARTICLE{Kuhn:136120,
author = {Kuhn, Peer-Hendrik and Wang, Huanhuan and Dislich, Bastian
and Colombo, Alessio and Zeitschel, Ulrike and Ellwart,
Joachim W and Kremmer, Elisabeth and Rossner, Steffen and
Lichtenthaler, Stefan},
title = {{ADAM}10 is the physiologically relevant, constitutive
alpha-secretase of the amyloid precursor protein in primary
neurons.},
journal = {The EMBO journal},
volume = {29},
number = {17},
issn = {0261-4189},
address = {Hoboken, NJ [u.a.]},
publisher = {Wiley},
reportid = {DZNE-2020-02442},
pages = {3020-3032},
year = {2010},
abstract = {The amyloid precursor protein (APP) undergoes constitutive
shedding by a protease activity called alpha-secretase. This
is considered an important mechanism preventing the
generation of the Alzheimer's disease amyloid-beta peptide
(Abeta). alpha-Secretase appears to be a metalloprotease of
the ADAM family, but its identity remains to be established.
Using a novel alpha-secretase-cleavage site-specific
antibody, we found that RNAi-mediated knockdown of ADAM10,
but surprisingly not of ADAM9 or 17, completely suppressed
APP alpha-secretase cleavage in different cell lines and in
primary murine neurons. Other proteases were not able to
compensate for this loss of alpha-cleavage. This finding was
further confirmed by mass-spectrometric detection of
APP-cleavage fragments. Surprisingly, in different cell
lines, the reduction of alpha-secretase cleavage was not
paralleled by a corresponding increase in the
Abeta-generating beta-secretase cleavage, revealing that
both proteases do not always compete for APP as a substrate.
Instead, our data suggest a novel pathway for APP
processing, in which ADAM10 can partially compete with
gamma-secretase for the cleavage of a C-terminal APP
fragment generated by beta-secretase. We conclude that
ADAM10 is the physiologically relevant, constitutive
alpha-secretase of APP.},
keywords = {ADAM Proteins: metabolism / ADAM10 Protein / Amyloid
Precursor Protein Secretases: metabolism / Amyloid
beta-Protein Precursor: metabolism / Animals / Cell Line /
Humans / Mass Spectrometry / Membrane Proteins: metabolism /
Mice / Neurons: enzymology / Neurons: metabolism / Amyloid
beta-Protein Precursor (NLM Chemicals) / Aplp1 protein,
mouse (NLM Chemicals) / Membrane Proteins (NLM Chemicals) /
Amyloid Precursor Protein Secretases (NLM Chemicals) / ADAM
Proteins (NLM Chemicals) / ADAM10 Protein (NLM Chemicals) /
Adam10 protein, mouse (NLM Chemicals)},
cin = {AG Lichtenthaler},
ddc = {570},
cid = {I:(DE-2719)1110006},
pnm = {342 - Disease Mechanisms and Model Systems (POF3-342)},
pid = {G:(DE-HGF)POF3-342},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:20676056},
pmc = {pmc:PMC2944055},
doi = {10.1038/emboj.2010.167},
url = {https://pub.dzne.de/record/136120},
}