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@ARTICLE{Zimmer:137739,
      author       = {Zimmer, Sebastian and Grebe, Alena and Latz, Eicke},
      title        = {{D}anger signaling in atherosclerosis.},
      journal      = {Circulation research},
      volume       = {116},
      number       = {2},
      issn         = {0009-7330},
      address      = {New York, NY},
      publisher    = {Assoc.},
      reportid     = {DZNE-2020-04061},
      pages        = {323-340},
      year         = {2015},
      abstract     = {All aspects of the pathogenesis of atherosclerosis are
                      critically influenced by the inflammatory response in
                      vascular plaques. Research in the field of innate immunity
                      from the past 2 decades has uncovered many novel mechanisms
                      elucidating how immune cells sense microbes, tissue damage,
                      and metabolic derangements. Here, we summarize which
                      triggers of innate immunity appear during atherogenesis and
                      by which pathways they can contribute to inflammation in
                      atherosclerotic plaques. The increased understanding gained
                      from studies assessing how immune activation is associated
                      with the pathogenesis of atherosclerosis has provided many
                      novel targets for potential therapeutic intervention.
                      Excitingly, the concept that inflammation may be the core of
                      cardiovascular disease is currently being clinically
                      evaluated and will probably encourage further studies in
                      this area.},
      subtyp        = {Review Article},
      keywords     = {Animals / Atherosclerosis: etiology / Atherosclerosis:
                      immunology / Atherosclerosis: microbiology / Humans /
                      Immunity, Innate: immunology / Signal Transduction:
                      immunology},
      cin          = {AG Latz},
      ddc          = {610},
      cid          = {I:(DE-2719)1013024},
      pnm          = {342 - Disease Mechanisms and Model Systems (POF3-342)},
      pid          = {G:(DE-HGF)POF3-342},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:25593277},
      doi          = {10.1161/CIRCRESAHA.116.301135},
      url          = {https://pub.dzne.de/record/137739},
}