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@ARTICLE{Zimmer:137739,
author = {Zimmer, Sebastian and Grebe, Alena and Latz, Eicke},
title = {{D}anger signaling in atherosclerosis.},
journal = {Circulation research},
volume = {116},
number = {2},
issn = {0009-7330},
address = {New York, NY},
publisher = {Assoc.},
reportid = {DZNE-2020-04061},
pages = {323-340},
year = {2015},
abstract = {All aspects of the pathogenesis of atherosclerosis are
critically influenced by the inflammatory response in
vascular plaques. Research in the field of innate immunity
from the past 2 decades has uncovered many novel mechanisms
elucidating how immune cells sense microbes, tissue damage,
and metabolic derangements. Here, we summarize which
triggers of innate immunity appear during atherogenesis and
by which pathways they can contribute to inflammation in
atherosclerotic plaques. The increased understanding gained
from studies assessing how immune activation is associated
with the pathogenesis of atherosclerosis has provided many
novel targets for potential therapeutic intervention.
Excitingly, the concept that inflammation may be the core of
cardiovascular disease is currently being clinically
evaluated and will probably encourage further studies in
this area.},
subtyp = {Review Article},
keywords = {Animals / Atherosclerosis: etiology / Atherosclerosis:
immunology / Atherosclerosis: microbiology / Humans /
Immunity, Innate: immunology / Signal Transduction:
immunology},
cin = {AG Latz},
ddc = {610},
cid = {I:(DE-2719)1013024},
pnm = {342 - Disease Mechanisms and Model Systems (POF3-342)},
pid = {G:(DE-HGF)POF3-342},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:25593277},
doi = {10.1161/CIRCRESAHA.116.301135},
url = {https://pub.dzne.de/record/137739},
}