000137851 001__ 137851 000137851 005__ 20240618103011.0 000137851 0247_ $$2doi$$a10.1016/S1474-4422(15)70016-5 000137851 0247_ $$2pmid$$apmid:25792098 000137851 0247_ $$2pmc$$apmc:PMC5909703 000137851 0247_ $$2ISSN$$a1474-4422 000137851 0247_ $$2ISSN$$a1474-4465 000137851 0247_ $$2altmetric$$aaltmetric:3808150 000137851 037__ $$aDZNE-2020-04173 000137851 041__ $$aEnglish 000137851 082__ $$a610 000137851 1001_ $$0P:(DE-2719)2000008$$aHeneka, Michael$$b0$$eFirst author 000137851 245__ $$aNeuroinflammation in Alzheimer's disease. 000137851 260__ $$aLondon$$bLancet Publ. Group$$c2015 000137851 264_1 $$2Crossref$$3print$$bElsevier BV$$c2015-04-01 000137851 3367_ $$2DRIVER$$aarticle 000137851 3367_ $$2DataCite$$aOutput Types/Journal article 000137851 3367_ $$0PUB:(DE-HGF)16$$2PUB:(DE-HGF)$$aJournal Article$$bjournal$$mjournal$$s1718699372_13420$$xReview Article 000137851 3367_ $$2BibTeX$$aARTICLE 000137851 3367_ $$2ORCID$$aJOURNAL_ARTICLE 000137851 3367_ $$00$$2EndNote$$aJournal Article 000137851 520__ $$aIncreasing evidence suggests that Alzheimer's disease pathogenesis is not restricted to the neuronal compartment, but includes strong interactions with immunological mechanisms in the brain. Misfolded and aggregated proteins bind to pattern recognition receptors on microglia and astroglia, and trigger an innate immune response characterised by release of inflammatory mediators, which contribute to disease progression and severity. Genome-wide analysis suggests that several genes that increase the risk for sporadic Alzheimer's disease encode factors that regulate glial clearance of misfolded proteins and the inflammatory reaction. External factors, including systemic inflammation and obesity, are likely to interfere with immunological processes of the brain and further promote disease progression. Modulation of risk factors and targeting of these immune mechanisms could lead to future therapeutic or preventive strategies for Alzheimer's disease. 000137851 536__ $$0G:(DE-HGF)POF3-342$$a342 - Disease Mechanisms and Model Systems (POF3-342)$$cPOF3-342$$fPOF III$$x0 000137851 536__ $$0G:(DE-HGF)POF3-344$$a344 - Clinical and Health Care Research (POF3-344)$$cPOF3-344$$fPOF III$$x1 000137851 542__ $$2Crossref$$i2015-04-01$$uhttps://www.elsevier.com/tdm/userlicense/1.0/ 000137851 588__ $$aDataset connected to CrossRef, PubMed, 000137851 650_7 $$2NLM Chemicals$$aAnti-Inflammatory Agents, Non-Steroidal 000137851 650_7 $$2NLM Chemicals$$aBiomarkers 000137851 650_7 $$2NLM Chemicals$$aInflammation Mediators 000137851 650_7 $$2NLM Chemicals$$aNootropic Agents 000137851 650_2 $$2MeSH$$aAlzheimer Disease: genetics 000137851 650_2 $$2MeSH$$aAlzheimer Disease: immunology 000137851 650_2 $$2MeSH$$aAlzheimer Disease: metabolism 000137851 650_2 $$2MeSH$$aAlzheimer Disease: pathology 000137851 650_2 $$2MeSH$$aAlzheimer Disease: prevention & control 000137851 650_2 $$2MeSH$$aAnimals 000137851 650_2 $$2MeSH$$aAnti-Inflammatory Agents, Non-Steroidal: therapeutic use 000137851 650_2 $$2MeSH$$aAstrocytes: immunology 000137851 650_2 $$2MeSH$$aAstrocytes: pathology 000137851 650_2 $$2MeSH$$aBiomarkers: blood 000137851 650_2 $$2MeSH$$aBiomarkers: cerebrospinal fluid 000137851 650_2 $$2MeSH$$aBrain Injuries: complications 000137851 650_2 $$2MeSH$$aBrain Injuries: metabolism 000137851 650_2 $$2MeSH$$aClinical Trials as Topic 000137851 650_2 $$2MeSH$$aDisease Models, Animal 000137851 650_2 $$2MeSH$$aDisease Progression 000137851 650_2 $$2MeSH$$aHumans 000137851 650_2 $$2MeSH$$aImmunity, Innate 000137851 650_2 $$2MeSH$$aImmunization 000137851 650_2 $$2MeSH$$aInflammation: diagnosis 000137851 650_2 $$2MeSH$$aInflammation: immunology 000137851 650_2 $$2MeSH$$aInflammation: metabolism 000137851 650_2 $$2MeSH$$aInflammation Mediators: immunology 000137851 650_2 $$2MeSH$$aInflammation Mediators: metabolism 000137851 650_2 $$2MeSH$$aLocus Coeruleus: pathology 000137851 650_2 $$2MeSH$$aMicroglia: immunology 000137851 650_2 $$2MeSH$$aMicroglia: pathology 000137851 650_2 $$2MeSH$$aNootropic Agents: administration & dosage 000137851 650_2 $$2MeSH$$aObesity: complications 000137851 650_2 $$2MeSH$$aObesity: metabolism 000137851 650_2 $$2MeSH$$aPhagocytosis 000137851 650_2 $$2MeSH$$aProtein Folding 000137851 650_2 $$2MeSH$$aRisk Factors 000137851 650_2 $$2MeSH$$aSeverity of Illness Index 000137851 7001_ $$aCarson, Monica J$$b1 000137851 7001_ $$aEl Khoury, Joseph$$b2 000137851 7001_ $$aLandreth, Gary E$$b3 000137851 7001_ $$0P:(DE-2719)2810593$$aBrosseron, Frederic$$b4 000137851 7001_ $$aFeinstein, Douglas L$$b5 000137851 7001_ $$aJacobs, Andreas H$$b6 000137851 7001_ $$aWyss-Coray, Tony$$b7 000137851 7001_ $$aVitorica, Javier$$b8 000137851 7001_ $$aRansohoff, Richard M$$b9 000137851 7001_ $$aHerrup, Karl$$b10 000137851 7001_ $$aFrautschy, Sally A$$b11 000137851 7001_ $$aFinsen, Bente$$b12 000137851 7001_ $$aBrown, Guy C$$b13 000137851 7001_ $$aVerkhratsky, Alexei$$b14 000137851 7001_ $$aYamanaka, Koji$$b15 000137851 7001_ $$aKoistinaho, Jari$$b16 000137851 7001_ $$0P:(DE-2719)2000062$$aLatz, Eicke$$b17 000137851 7001_ $$0P:(DE-HGF)0$$aHalle, Annett$$b18 000137851 7001_ $$0P:(DE-2719)2810273$$aPetzold, Gabor C$$b19 000137851 7001_ $$aTown, Terrence$$b20 000137851 7001_ $$aMorgan, Dave$$b21 000137851 7001_ $$aShinohara, Mari L$$b22 000137851 7001_ $$aPerry, V Hugh$$b23 000137851 7001_ $$aHolmes, Clive$$b24 000137851 7001_ $$aBazan, Nicolas G$$b25 000137851 7001_ $$aBrooks, David J$$b26 000137851 7001_ $$aHunot, Stéphane$$b27 000137851 7001_ $$aJoseph, Bertrand$$b28 000137851 7001_ $$aDeigendesch, Nikolaus$$b29 000137851 7001_ $$aGaraschuk, Olga$$b30 000137851 7001_ $$aBoddeke, Erik$$b31 000137851 7001_ $$aDinarello, Charles A$$b32 000137851 7001_ $$aBreitner, John C$$b33 000137851 7001_ $$aCole, Greg M$$b34 000137851 7001_ $$aGolenbock, Douglas T$$b35 000137851 7001_ $$0P:(DE-HGF)0$$aKummer, Markus P$$b36 000137851 77318 $$2Crossref$$3journal-article$$a10.1016/s1474-4422(15)70016-5$$b : Elsevier BV, 2015-04-01$$n4$$p388-405$$tThe Lancet Neurology$$v14$$x1474-4422$$y2015 000137851 773__ $$0PERI:(DE-600)2079704-7$$a10.1016/S1474-4422(15)70016-5$$gVol. 14, no. 4, p. 388 - 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