Home > Publications Database > Pharmacological treatment options for mast cell activation disease. > print

001     138621
005     20240411115838.0
024 7 _ |a 0365-2009
|2 ISSN
024 7 _ |a 10.1007/s00210-016-1247-1
|2 doi
024 7 _ |a pmid:27132234
|2 pmid
024 7 _ |a pmc:PMC4903110
|2 pmc
024 7 _ |a 0028-1298
|2 ISSN
024 7 _ |a 1432-1912
|2 ISSN
024 7 _ |a altmetric:7111016
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024 7 _ |a 0365-5423
|2 ISSN
024 7 _ |a 0340-5249
|2 ISSN
037 _ _ |a DZNE-2020-04943
041 _ _ |a English
082 _ _ |a 610
100 1 _ |a Molderings, Gerhard J
|0 P:(DE-HGF)0
|b 0
|e Corresponding author
245 _ _ |a Pharmacological treatment options for mast cell activation disease.
260 _ _ |a Heidelberg
|c 2016
|b Springer
264 _ 1 |3 online
|2 Crossref
|b Springer Science and Business Media LLC
|c 2016-04-30
264 _ 1 |3 print
|2 Crossref
|b Springer Science and Business Media LLC
|c 2016-07-01
336 7 _ |a article
|2 DRIVER
336 7 _ |a Output Types/Journal article
|2 DataCite
336 7 _ |a Journal Article
|b journal
|m journal
|0 PUB:(DE-HGF)16
|s 1712748537_11352
|2 PUB:(DE-HGF)
|x Review Article
336 7 _ |a ARTICLE
|2 BibTeX
336 7 _ |a JOURNAL_ARTICLE
|2 ORCID
336 7 _ |a Journal Article
|0 0
|2 EndNote
520 _ _ |a Mast cell activation disease (MCAD) is a term referring to a heterogeneous group of disorders characterized by aberrant release of variable subsets of mast cell (MC) mediators together with accumulation of either morphologically altered and immunohistochemically identifiable mutated MCs due to MC proliferation (systemic mastocytosis [SM] and MC leukemia [MCL]) or morphologically ordinary MCs due to decreased apoptosis (MC activation syndrome [MCAS] and well-differentiated SM). Clinical signs and symptoms in MCAD vary depending on disease subtype and result from excessive mediator release by MCs and, in aggressive forms, from organ failure related to MC infiltration. In most cases, treatment of MCAD is directed primarily at controlling the symptoms associated with MC mediator release. In advanced forms, such as aggressive SM and MCL, agents targeting MC proliferation such as kinase inhibitors may be provided. Targeted therapies aimed at blocking mutant protein variants and/or downstream signaling pathways are currently being developed. Other targets, such as specific surface antigens expressed on neoplastic MCs, might be considered for the development of future therapies. Since clinicians are often underprepared to evaluate, diagnose, and effectively treat this clinically heterogeneous disease, we seek to familiarize clinicians with MCAD and review current and future treatment approaches.
536 _ _ |a 345 - Population Studies and Genetics (POF3-345)
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|c POF3-345
|f POF III
|x 0
542 _ _ |i 2016-04-30
|2 Crossref
|u http://creativecommons.org/licenses/by/4.0
588 _ _ |a Dataset connected to CrossRef, PubMed,
650 _ 7 |a Antineoplastic Agents
|2 NLM Chemicals
650 _ 7 |a Histamine Antagonists
|2 NLM Chemicals
650 _ 7 |a Immunosuppressive Agents
|2 NLM Chemicals
650 _ 2 |a Animals
|2 MeSH
650 _ 2 |a Antineoplastic Agents: adverse effects
|2 MeSH
650 _ 2 |a Antineoplastic Agents: therapeutic use
|2 MeSH
650 _ 2 |a Apoptosis
|2 MeSH
650 _ 2 |a Cell Degranulation: drug effects
|2 MeSH
650 _ 2 |a Cell Proliferation: drug effects
|2 MeSH
650 _ 2 |a Histamine Antagonists: adverse effects
|2 MeSH
650 _ 2 |a Histamine Antagonists: therapeutic use
|2 MeSH
650 _ 2 |a Humans
|2 MeSH
650 _ 2 |a Immunosuppressive Agents: adverse effects
|2 MeSH
650 _ 2 |a Immunosuppressive Agents: therapeutic use
|2 MeSH
650 _ 2 |a Leukemia, Mast-Cell: drug therapy
|2 MeSH
650 _ 2 |a Leukemia, Mast-Cell: immunology
|2 MeSH
650 _ 2 |a Leukemia, Mast-Cell: metabolism
|2 MeSH
650 _ 2 |a Leukemia, Mast-Cell: pathology
|2 MeSH
650 _ 2 |a Mast Cells: drug effects
|2 MeSH
650 _ 2 |a Mast Cells: immunology
|2 MeSH
650 _ 2 |a Mast Cells: metabolism
|2 MeSH
650 _ 2 |a Mast Cells: pathology
|2 MeSH
650 _ 2 |a Mastocytosis, Systemic: drug therapy
|2 MeSH
650 _ 2 |a Mastocytosis, Systemic: immunology
|2 MeSH
650 _ 2 |a Mastocytosis, Systemic: metabolism
|2 MeSH
650 _ 2 |a Mastocytosis, Systemic: pathology
|2 MeSH
650 _ 2 |a Molecular Targeted Therapy
|2 MeSH
650 _ 2 |a Treatment Outcome
|2 MeSH
700 1 _ |a Haenisch, Britta
|0 P:(DE-2719)2810511
|b 1
|u dzne
700 1 _ |a Brettner, Stefan
|b 2
700 1 _ |a Homann, Jürgen
|b 3
700 1 _ |a Menzen, Markus
|b 4
700 1 _ |a Dumoulin, Franz Ludwig
|b 5
700 1 _ |a Panse, Jens
|b 6
700 1 _ |a Butterfield, Joseph
|b 7
700 1 _ |a Afrin, Lawrence B
|b 8
773 1 8 |a 10.1007/s00210-016-1247-1
|b : Springer Science and Business Media LLC, 2016-04-30
|n 7
|p 671-694
|3 journal-article
|2 Crossref
|t Naunyn-Schmiedeberg's Archives of Pharmacology
|v 389
|y 2016
|x 0028-1298
773 _ _ |a 10.1007/s00210-016-1247-1
|g Vol. 389, no. 7, p. 671 - 694
|0 PERI:(DE-600)1462940-9
|n 7
|q 389:7<671 - 694
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|t Naunyn-Schmiedeberg's archives of pharmacology
|v 389
|y 2016
|x 0028-1298
856 4 _ |y OpenAccess
|u https://pub.dzne.de/record/138621/files/DZNE-2020-04943.pdf
856 4 _ |y OpenAccess
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856 7 _ |2 Pubmed Central
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910 1 _ |a Deutsches Zentrum für Neurodegenerative Erkrankungen
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913 1 _ |a DE-HGF
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LibraryCollectionCLSMajorCLSMinorLanguageAuthor
Marc 21