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@ARTICLE{Louwersheimer:138696,
author = {Louwersheimer, Eva and Wolfsgruber, Steffen and Espinosa,
Ana and Lacour, André and Heilmann-Heimbach, Stefanie and
Alegret, Montserrat and Hernández, Isabel and Rosende-Roca,
Maitée and Tárraga, Lluís and Boada, Mercè and
Kornhuber, Johannes and Peters, Oliver and Frölich, Lutz
and Hüll, Michael and Rüther, Eckart and Wiltfang, Jens
and Scherer, Martin and Riedel-Heller, Steffi and Jessen,
Frank and Nöthen, Markus M and Maier, Wolfgang and Koene,
Ted and Scheltens, Philip and Holstege, Henne and Wagner,
Michael and Ruiz, Agustín and van der Flier, Wiesje M and
Becker, Tim and Ramirez, Alfredo},
title = {{A}lzheimer's disease risk variants modulate endophenotypes
in mild cognitive impairment.},
journal = {Alzheimer's and dementia},
volume = {12},
number = {8},
issn = {1552-5260},
address = {Amsterdam [u.a.]},
publisher = {Elsevier},
reportid = {DZNE-2020-05018},
pages = {872-881},
year = {2016},
abstract = {We evaluated the effect of Alzheimer's disease (AD)
susceptibility loci on endophenotypes closely related with
AD pathology in patients with mild cognitive impairment
(MCI).We selected 1730 MCI patients from four independent
data sets. Weighted polygenic risk scores (PGS) were
constructed of 18 non-apolipoprotein E (APOE) AD risk
variants. In addition, we determined APOE genotype. AD
endophenotypes were cognitive decline over time and
cerebrospinal fluid (CSF) biomarkers (aβ, tau, ptau).PGS
was modestly associated with cognitive decline over time, as
measured by mini-mental state examination (MMSE) (β ±
SE:-0.24 ± 0.10; P = .012), and with CSF levels of tau
and ptau (tau: 1.38 ± 0.36, P = 1.21 × 10(-4); ptau:
1.40 ± 0.36, P = 1.02 × 10(-4)).In MCI, we observed a
joint effect of AD susceptibility loci on nonamyloid
endophenotypes, suggesting a link of these genetic loci with
neuronal degeneration in general rather than with
Alzheimer-related amyloid deposition.},
keywords = {Aged / Aged, 80 and over / Alzheimer Disease: cerebrospinal
fluid / Alzheimer Disease: diagnosis / Amyloid
beta-Peptides: cerebrospinal fluid / Apolipoproteins E:
genetics / Cognitive Dysfunction: cerebrospinal fluid /
Cognitive Dysfunction: complications / Cognitive
Dysfunction: genetics / Cohort Studies / Datasets as Topic:
statistics $\&$ numerical data / Endophenotypes:
cerebrospinal fluid / Female / Humans / Male / Mental Status
Schedule / Middle Aged / Multifactorial Inheritance:
genetics / Neuropsychological Tests / Odds Ratio /
Polymorphism, Single Nucleotide: genetics / Statistics,
Nonparametric / Amyloid beta-Peptides (NLM Chemicals) /
Apolipoproteins E (NLM Chemicals)},
cin = {AG Wagner / AG Becker ; GenomMathematik ; GenomMathematik /
U Clinical Researchers - Bonn},
ddc = {610},
cid = {I:(DE-2719)1011201 / I:(DE-2719)1013007 /
I:(DE-2719)7000001},
pnm = {344 - Clinical and Health Care Research (POF3-344) / 345 -
Population Studies and Genetics (POF3-345)},
pid = {G:(DE-HGF)POF3-344 / G:(DE-HGF)POF3-345},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:26921674},
doi = {10.1016/j.jalz.2016.01.006},
url = {https://pub.dzne.de/record/138696},
}