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@ARTICLE{Mentrup:139570,
author = {Mentrup, Torben and Loock, Ann-Christine and Fluhrer,
Regina and Schröder, Bernd},
title = {{S}ignal peptide peptidase and {SPP}-like proteases -
{P}ossible therapeutic targets?},
journal = {Biochimica et biophysica acta / Molecular cell research},
volume = {1864},
number = {11},
issn = {0167-4889},
address = {Amsterdam [u.a.]},
publisher = {Elsevier},
reportid = {DZNE-2020-05892},
pages = {2169-2182},
year = {2017},
abstract = {Signal peptide peptidase (SPP) and the four homologous
SPP-like proteases SPPL2a, SPPL2b, SPPL2c and SPPL3 are
GxGD-type intramembrane-cleaving proteases (I-CLIPs). In
addition to divergent subcellular localisations, distinct
differences in the mechanistic properties and substrate
requirements of individual family members have been
unravelled. SPP/SPPL proteases employ a catalytic mechanism
related to that of the γ-secretase complex. Nevertheless,
differential targeting of SPP/SPPL proteases and
γ-secretase by inhibitors has been demonstrated.
Furthermore, also within the SPP/SPPL family significant
differences in the sensitivity to currently available
inhibitory compounds have been reported. Though far from
complete, our knowledge on pathophysiological functions of
SPP/SPPL proteases, in particular based on studies in mice,
has been significantly increased over the last years. Based
on this, inhibition of distinct SPP/SPPL proteases has been
proposed as a novel therapeutic concept e.g. for the
treatment of autoimmunity and viral or protozoal infections,
as we will discuss in this review. This article is part of a
Special Issue entitled: Proteolysis as a Regulatory Event in
Pathophysiology edited by Stefan Rose-John.},
subtyp = {Review Article},
keywords = {Amino Acid Sequence: genetics / Amyloid Precursor Protein
Secretases: antagonists $\&$ inhibitors / Amyloid Precursor
Protein Secretases: genetics / Aspartic Acid Endopeptidases:
antagonists $\&$ inhibitors / Aspartic Acid Endopeptidases:
genetics / Humans / Membrane Proteins: antagonists $\&$
inhibitors / Membrane Proteins: genetics / Peptides:
antagonists $\&$ inhibitors / Peptides: genetics / Peptides:
metabolism / Proteolysis / Substrate Specificity / Membrane
Proteins (NLM Chemicals) / Peptides (NLM Chemicals) /
dermcidin (NLM Chemicals) / Amyloid Precursor Protein
Secretases (NLM Chemicals) / Aspartic Acid Endopeptidases
(NLM Chemicals) / SPPL2a protein, human (NLM Chemicals) /
SPPL2b protein, human (NLM Chemicals)},
cin = {AG Fluhrer},
ddc = {570},
cid = {I:(DE-2719)1110000-2},
pnm = {342 - Disease Mechanisms and Model Systems (POF3-342)},
pid = {G:(DE-HGF)POF3-342},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:28624439},
doi = {10.1016/j.bbamcr.2017.06.007},
url = {https://pub.dzne.de/record/139570},
}