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@ARTICLE{Ziegler:140092,
author = {Ziegler, Gabriel and Grabher, Patrick and Thompson, Alan
and Altmann, Daniel and Hupp, Markus and Ashburner, John and
Friston, Karl and Weiskopf, Nikolaus and Curt, Armin and
Freund, Patrick},
title = {{P}rogressive neurodegeneration following spinal cord
injury: {I}mplications for clinical trials.},
journal = {Neurology},
volume = {90},
number = {14},
issn = {0028-3878},
address = {[S.l.]},
publisher = {Ovid},
reportid = {DZNE-2020-06414},
pages = {e1257-e1266},
year = {2018},
abstract = {To quantify atrophy, demyelination, and iron accumulation
over 2 years following acute spinal cord injury and to
identify MRI predictors of clinical outcomes and determine
their suitability as surrogate markers of therapeutic
intervention.We assessed 156 quantitative MRI datasets from
15 patients with spinal cord injury and 18 controls at
baseline and 2, 6, 12, and 24 months after injury. Clinical
recovery (including neuropathic pain) was assessed at each
time point. Between-group differences in linear and
nonlinear trajectories of volume, myelin, and iron change
were estimated. Structural changes by 6 months were used to
predict clinical outcomes at 2 years.The majority of
patients showed clinical improvement with recovery
stabilizing at 2 years. Cord atrophy decelerated, while
cortical white and gray matter atrophy progressed over 2
years. Myelin content in the spinal cord and cortex
decreased progressively over time, while cerebellar loss
decreases decelerated. As atrophy progressed in the
thalamus, sustained iron accumulation was evident. Smaller
cord and cranial corticospinal tract atrophy, and myelin
changes within the sensorimotor cortices, by 6 months
predicted recovery in lower extremity motor score at 2
years. Whereas greater cord atrophy and microstructural
changes in the cerebellum, anterior cingulate cortex, and
secondary sensory cortex by 6 months predicted worse sensory
impairment and greater neuropathic pain intensity at 2
years.These results draw attention to trauma-induced
neuroplastic processes and highlight the intimate
relationships among neurodegenerative processes in the cord
and brain. These measurable changes are sufficiently large,
systematic, and predictive to render them viable outcome
measures for clinical trials.},
keywords = {Adult / Aged / Atrophy / Brain: diagnostic imaging / Brain:
pathology / Disease Progression / Female / Follow-Up Studies
/ Humans / Longitudinal Studies / Male / Middle Aged / Nerve
Degeneration: diagnostic imaging / Nerve Degeneration:
etiology / Nerve Degeneration: physiopathology / Neuralgia:
diagnostic imaging / Neuralgia: etiology / Neuralgia:
physiopathology / Neurodegenerative Diseases: diagnostic
imaging / Neurodegenerative Diseases: etiology /
Neurodegenerative Diseases: physiopathology / Recovery of
Function / Spinal Cord: diagnostic imaging / Spinal Cord:
pathology / Spinal Cord Injuries: complications / Spinal
Cord Injuries: diagnostic imaging / Spinal Cord Injuries:
physiopathology / Young Adult},
cin = {AG Düzel},
ddc = {610},
cid = {I:(DE-2719)5000006},
pnm = {344 - Clinical and Health Care Research (POF3-344)},
pid = {G:(DE-HGF)POF3-344},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:29514946},
pmc = {pmc:PMC5890610},
doi = {10.1212/WNL.0000000000005258},
url = {https://pub.dzne.de/record/140092},
}