TY - JOUR
AU - Wüsten, Annick
AU - Pasham Parameshwar, Reddy
AU - Smiyakin, Andrej
AU - Bernis, Maria Eugenia
AU - Tamgüney, Gültekin
TI - A Bioluminescent Cell Assay to Quantify Prion Protein Dimerization.
JO - Scientific reports
VL - 8
IS - 1
SN - 2045-2322
CY - [London]
PB - Macmillan Publishers Limited, part of Springer Nature
M1 - DZNE-2020-06536
SP - 14178
PY - 2018
AB - The prion protein (PrP) is a cell surface protein that in disease misfolds and becomes infectious causing Creutzfeldt-Jakob disease in humans, scrapie in sheep, and chronic wasting disease in deer and elk. Little is known regarding the dimerization of PrP and its role in disease. We developed a bioluminescent prion assay (BPA) to quantify PrP dimerization by bimolecular complementation of split Gaussia luciferase (GLuc) halves that are each fused to PrP. Fusion constructs between PrP and N- and C-terminal GLuc halves were expressed on the surface of RK13 cells (RK13-DC cells) and dimerized to yield a bioluminescent signal that was decreased in the presence of eight different antibodies to PrP. Dimerization of PrP was independent of divalent cations and was induced under stress. Challenge of RK13-DC cells with seven different prion strains did not lead to detectable infection but was measurable by bioluminescence. Finally, we used BPA to screen a compound library for compounds inhibiting PrP dimerization. One of the most potent compounds to inhibit PrP dimerization was JTC-801, which also inhibited prion replication in RML-infected ScN2a and SMB cells with an EC50 of 370 nM and 220 nM, respectively. We show here that BPA is a versatile tool to study prion biology and to identify anti-prion compounds.
KW - Animals
KW - Biological Assay: methods
KW - Cations, Divalent: metabolism
KW - Cell Line
KW - Cell Line, Tumor
KW - Creutzfeldt-Jakob Syndrome: metabolism
KW - Deer
KW - Dimerization
KW - Humans
KW - Luminescent Measurements: methods
KW - Mice
KW - Prion Proteins: metabolism
KW - Protein Folding
KW - Rabbits
KW - Scrapie: metabolism
KW - Sheep
KW - Wasting Disease, Chronic: metabolism
KW - Cations, Divalent (NLM Chemicals)
KW - Prion Proteins (NLM Chemicals)
LB - PUB:(DE-HGF)16
C6 - pmid:30242186
C2 - pmc:PMC6155003
DO - DOI:10.1038/s41598-018-32581-1
UR - https://pub.dzne.de/record/140214
ER -