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000140256 0247_ $$2doi$$a10.1007/s11910-018-0898-3
000140256 0247_ $$2pmid$$apmid:30293094
000140256 0247_ $$2ISSN$$a1528-4042
000140256 0247_ $$2ISSN$$a1534-6293
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000140256 037__ $$aDZNE-2020-06578
000140256 041__ $$aEnglish
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000140256 1001_ $$0P:(DE-HGF)0$$aHammes, Jochen$$b0$$eCorresponding author
000140256 245__ $$aThe Role of Tau Imaging in Parkinsonian Disorders.
000140256 260__ $$aPhiladelphia, Pa.$$bCurrent Science Inc.$$c2018
000140256 264_1 $$2Crossref$$3online$$bSpringer Science and Business Media LLC$$c2018-10-06
000140256 264_1 $$2Crossref$$3print$$bSpringer Science and Business Media LLC$$c2018-12-01
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000140256 520__ $$aDifferential diagnosis of atypical Parkinson syndromes (APS) is difficult as clinical presentations may vary and as there is a strong overlap between disease entities. Aggregations of misfolded and hyperphosphorylated tau proteins are the common denominator of many of these diseases.Several tau targeting positron emission tomography (PET) tracers have been evaluated as possible biomarkers in APS in the recent years. For Parkinson's disease, dementia with Lewy bodies, progressive supranuclear palsy, and corticobasal degeneration, promising results have been reported with regard to the ability to detect the presence of disease and to discriminate patients from controls. However, the discussion about the specificity of the first-generation radiotracers and their value in the clinical context is ongoing. A combined interpretation of signal strength and distribution pattern in PET scans with first- and second-generation tracers may be helpful in clinical diagnosis and follow-up of patients with APS.
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000140256 650_7 $$2NLM Chemicals$$atau Proteins
000140256 650_2 $$2MeSH$$aAlzheimer Disease: diagnosis
000140256 650_2 $$2MeSH$$aAlzheimer Disease: diagnostic imaging
000140256 650_2 $$2MeSH$$aAlzheimer Disease: metabolism
000140256 650_2 $$2MeSH$$aDiagnosis, Differential
000140256 650_2 $$2MeSH$$aHumans
000140256 650_2 $$2MeSH$$aParkinsonian Disorders: diagnostic imaging
000140256 650_2 $$2MeSH$$aParkinsonian Disorders: metabolism
000140256 650_2 $$2MeSH$$aPositron-Emission Tomography: methods
000140256 650_2 $$2MeSH$$aSupranuclear Palsy, Progressive: diagnosis
000140256 650_2 $$2MeSH$$aSupranuclear Palsy, Progressive: diagnostic imaging
000140256 650_2 $$2MeSH$$aSupranuclear Palsy, Progressive: metabolism
000140256 650_2 $$2MeSH$$atau Proteins: metabolism
000140256 7001_ $$0P:(DE-2719)2811239$$aDrzezga, Alexander$$b1$$udzne
000140256 7001_ $$0P:(DE-2719)2812285$$aEimeren, Thilo$$b2$$eLast author$$udzne
000140256 77318 $$2Crossref$$3journal-article$$a10.1007/s11910-018-0898-3$$b : Springer Science and Business Media LLC, 2018-10-06$$n12$$p86$$tCurrent Neurology and Neuroscience Reports$$v18$$x1528-4042$$y2018
000140256 773__ $$0PERI:(DE-600)2094171-7$$a10.1007/s11910-018-0898-3$$gVol. 18, no. 12, p. 86$$n12$$p86$$q18:12<86$$tCurrent neurology and neuroscience reports$$v18$$x1528-4042$$y2018
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000140256 9141_ $$y2018
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