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@ARTICLE{Hans:140270,
author = {Hans, Friederike and Eckert, Marita and von Zweydorf, Felix
and Gloeckner, Christian Johannes and Kahle, Philipp J},
title = {{I}dentification and characterization of ubiquitinylation
sites in {TAR} {DNA}-binding protein of 43 k{D}a
({TDP}-43).},
journal = {The journal of biological chemistry},
volume = {293},
number = {41},
issn = {0021-9258},
address = {Bethesda, Md.},
publisher = {Soc.60645},
reportid = {DZNE-2020-06592},
pages = {16083-16099},
year = {2018},
abstract = {TAR DNA-binding protein of 43 kDa (TDP-43) forms
pathological aggregates in neurodegenerative diseases,
particularly in certain forms of frontotemporal dementia and
amyotrophic lateral sclerosis. Pathological modifications of
TDP-43 include proteolytic fragmentation, phosphorylation,
and ubiquitinylation. A pathognomonic TDP-43 C-terminal
fragment (CTF) spanning amino acids 193-414 contains only
four lysine residues that could be potentially
ubiquitinylated. Here, serial mutagenesis of these four
lysines to arginine revealed that not a single residue is
responsible for the ubiquitinylation of mCherry-tagged CTF.
Removal of all four lysines was necessary to suppress
ubiquitinylation. Interestingly, Lys-408 substitution
enhanced the pathological phosphorylation of the immediately
adjacent serine residues 409/410 in the context of
mCherry-CTF. Thus, Lys-408 ubiquitinylation appears to
hinder Ser-409/410 phosphorylation in TDP-43 CTF. However,
we did not observe the same effect for full-length TDP-43.
We extended the mutagenesis study to full-length TDP-43 and
performed MS. Ubiquitinylated lysine residues were
identified in the nuclear localization sequence (NLS; Lys-84
and Lys-95) and RNA-binding region (mostly Lys-160, Lys-181,
and Lys-263). Mutagenesis of Lys-84 confirmed its importance
as the major determinant for nuclear import, whereas Lys-95
mutagenesis did not significantly affect TDP-43's
nucleo-cytoplasmic distribution, solubility, aggregation,
and RNA-processing activities. Nevertheless, the K95A mutant
had significantly reduced Ser-409/410 phosphorylation,
emphasizing the suspected interplay between TDP-43
ubiquitinylation and phosphorylation. Collectively, our
analysis of TDP-43 ubiquitinylation sites indicates that the
NLS residues Lys-84 and Lys-95 have more prominent roles in
TDP-43 function than the more C-terminal lysines and
suggests a link between specific ubiquitinylation events and
pathological TDP-43 phosphorylation.},
keywords = {Active Transport, Cell Nucleus / Cell Nucleus: metabolism /
DNA-Binding Proteins: chemistry / DNA-Binding Proteins:
genetics / DNA-Binding Proteins: metabolism / HEK293 Cells /
Humans / Lysine: chemistry / Mutagenesis, Site-Directed /
Mutation / Phosphorylation / Protein Processing,
Post-Translational / Solubility / Ubiquitin: metabolism /
DNA-Binding Proteins (NLM Chemicals) / TDP-43 protein, human
(NLM Chemicals) / Ubiquitin (NLM Chemicals) / Lysine (NLM
Chemicals)},
cin = {AG Kahle / AG Gloeckner},
ddc = {540},
cid = {I:(DE-2719)1210000-4 / I:(DE-2719)1210007},
pnm = {345 - Population Studies and Genetics (POF3-345)},
pid = {G:(DE-HGF)POF3-345},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:30120199},
pmc = {pmc:PMC6187624},
doi = {10.1074/jbc.RA118.003440},
url = {https://pub.dzne.de/record/140270},
}