TY - JOUR AU - van der Lee, Sven J AU - Conway, Olivia J AU - Jansen, Iris AU - Carrasquillo, Minerva M AU - Kleineidam, Luca AU - van den Akker, Erik AU - Hernández, Isabel AU - van Eijk, Kristel R AU - Stringa, Najada AU - Chen, Jason A AU - Zettergren, Anna AU - Andlauer, Till F M AU - Diez-Fairen, Monica AU - Simon Sanchez, Javier AU - Lleó, Alberto AU - Zetterberg, Henrik AU - Nygaard, Marianne AU - Blauwendraat, Cornelis AU - Savage, Jeanne E AU - Mengel-From, Jonas AU - Moreno-Grau, Sonia AU - Wagner, Michael AU - Fortea, Juan AU - Keogh, Michael J AU - Blennow, Kaj AU - Skoog, Ingmar AU - Friese, Manuel A AU - Pletnikova, Olga AU - Zulaica, Miren AU - Lage, Carmen AU - de Rojas, Itziar AU - Riedel-Heller, Steffi AU - Illán-Gala, Ignacio AU - Wei, Wei AU - Jeune, Bernard AU - Orellana, Adelina AU - Then Bergh, Florian AU - Wang, Xue AU - Hulsman, Marc AU - Beker, Nina AU - Tesi, Niccolo AU - Morris, Christopher M AU - Indakoetxea, Begoña AU - Collij, Lyduine E AU - Scherer, Martin AU - Morenas-Rodríguez, Estrella AU - Ironside, James W AU - van Berckel, Bart N M AU - Alcolea, Daniel AU - Wiendl, Heinz AU - Strickland, Samantha L AU - Pastor, Pau AU - Rodríguez Rodríguez, Eloy AU - DESGESCO AU - EADB AU - IFGC AU - IPDGC AU - RiMod-FTD AU - Bank, Netherlands Brain AU - Boeve, Bradley F AU - Petersen, Ronald C AU - Ferman, Tanis J AU - van Gerpen, Jay A AU - Reinders, Marcel J T AU - Uitti, Ryan J AU - Tárraga, Lluís AU - Maier, Wolfgang AU - Dols-Icardo, Oriol AU - Kawalia, Amit AU - Dalmasso, Maria Carolina AU - Boada, Mercè AU - Zettl, Uwe K AU - van Schoor, Natasja M AU - Beekman, Marian AU - Allen, Mariet AU - Masliah, Eliezer AU - de Munain, Adolfo López AU - Pantelyat, Alexander AU - Wszolek, Zbigniew K AU - Ross, Owen A AU - Dickson, Dennis W AU - Graff-Radford, Neill R AU - Knopman, David AU - Rademakers, Rosa AU - Lemstra, Afina W AU - Pijnenburg, Yolande A L AU - Scheltens, Philip AU - Gasser, Thomas AU - Chinnery, Patrick F AU - Hemmer, Bernhard AU - Huisman, Martijn A AU - Troncoso, Juan AU - Moreno, Fermin AU - Nohr, Ellen A AU - Sørensen, Thorkild I A AU - Heutink, Peter AU - Sánchez-Juan, Pascual AU - Posthuma, Danielle AU - GIFT AU - Clarimón, Jordi AU - Christensen, Kaare AU - Ertekin-Taner, Nilüfer AU - Scholz, Sonja W AU - Ramirez, Alfredo AU - Ruiz, Agustín AU - Slagboom, Eline AU - van der Flier, Wiesje M AU - Holstege, Henne AU - Coppola, G. AU - Karydas, A. M. AU - Varpetian, A. AU - Foroud, T. M. AU - Levey, A. I. AU - Kukull, W. A. AU - Mendez, M. F. AU - Ringman, J. AU - Chui, H. AU - Cotman, C. AU - DeCarli, C. AU - Miller, B. L. AU - Geschwind, D. H. TI - A nonsynonymous mutation in PLCG2 reduces the risk of Alzheimer's disease, dementia with Lewy bodies and frontotemporal dementia, and increases the likelihood of longevity. JO - Acta neuropathologica VL - 138 IS - 2 SN - 0001-6322 CY - Heidelberg PB - Springer M1 - DZNE-2020-07162 SP - 237-250 PY - 2019 AB - The genetic variant rs72824905-G (minor allele) in the PLCG2 gene was previously associated with a reduced Alzheimer's disease risk (AD). The role of PLCG2 in immune system signaling suggests it may also protect against other neurodegenerative diseases and possibly associates with longevity. We studied the effect of the rs72824905-G on seven neurodegenerative diseases and longevity, using 53,627 patients, 3,516 long-lived individuals and 149,290 study-matched controls. We replicated the association of rs72824905-G with reduced AD risk and we found an association with reduced risk of dementia with Lewy bodies (DLB) and frontotemporal dementia (FTD). We did not find evidence for an effect on Parkinson's disease (PD), amyotrophic lateral sclerosis (ALS) and multiple sclerosis (MS) risks, despite adequate sample sizes. Conversely, the rs72824905-G allele was associated with increased likelihood of longevity. By-proxy analyses in the UK Biobank supported the associations with both dementia and longevity. Concluding, rs72824905-G has a protective effect against multiple neurodegenerative diseases indicating shared aspects of disease etiology. Our findings merit studying the PLCγ2 pathway as drug-target. KW - Alleles KW - Alzheimer Disease: genetics KW - Amyotrophic Lateral Sclerosis: genetics KW - Brain: immunology KW - Brain: metabolism KW - Brain: pathology KW - Dementia: genetics KW - Frontotemporal Dementia: genetics KW - Genetic Predisposition to Disease KW - Genome-Wide Association Study KW - Humans KW - Lewy Body Disease: genetics KW - Longevity: genetics KW - Microglia: metabolism KW - Multiple Sclerosis: genetics KW - Mutation KW - Neuroimaging KW - Parkinson Disease: genetics KW - Phospholipase C gamma: genetics KW - Risk LB - PUB:(DE-HGF)16 C6 - pmid:31131421 C2 - pmc:PMC6660501 DO - DOI:10.1007/s00401-019-02026-8 UR - https://pub.dzne.de/record/140840 ER -