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@ARTICLE{vanderLee:140840,
author = {van der Lee, Sven J and Conway, Olivia J and Jansen, Iris
and Carrasquillo, Minerva M and Kleineidam, Luca and van den
Akker, Erik and Hernández, Isabel and van Eijk, Kristel R
and Stringa, Najada and Chen, Jason A and Zettergren, Anna
and Andlauer, Till F M and Diez-Fairen, Monica and Simon
Sanchez, Javier and Lleó, Alberto and Zetterberg, Henrik
and Nygaard, Marianne and Blauwendraat, Cornelis and Savage,
Jeanne E and Mengel-From, Jonas and Moreno-Grau, Sonia and
Wagner, Michael and Fortea, Juan and Keogh, Michael J and
Blennow, Kaj and Skoog, Ingmar and Friese, Manuel A and
Pletnikova, Olga and Zulaica, Miren and Lage, Carmen and de
Rojas, Itziar and Riedel-Heller, Steffi and Illán-Gala,
Ignacio and Wei, Wei and Jeune, Bernard and Orellana,
Adelina and Then Bergh, Florian and Wang, Xue and Hulsman,
Marc and Beker, Nina and Tesi, Niccolo and Morris,
Christopher M and Indakoetxea, Begoña and Collij, Lyduine E
and Scherer, Martin and Morenas-Rodríguez, Estrella and
Ironside, James W and van Berckel, Bart N M and Alcolea,
Daniel and Wiendl, Heinz and Strickland, Samantha L and
Pastor, Pau and Rodríguez Rodríguez, Eloy and DESGESCO and
EADB and IFGC and IPDGC and RiMod-FTD and Bank, Netherlands
Brain and Boeve, Bradley F and Petersen, Ronald C and
Ferman, Tanis J and van Gerpen, Jay A and Reinders, Marcel J
T and Uitti, Ryan J and Tárraga, Lluís and Maier, Wolfgang
and Dols-Icardo, Oriol and Kawalia, Amit and Dalmasso, Maria
Carolina and Boada, Mercè and Zettl, Uwe K and van Schoor,
Natasja M and Beekman, Marian and Allen, Mariet and Masliah,
Eliezer and de Munain, Adolfo López and Pantelyat,
Alexander and Wszolek, Zbigniew K and Ross, Owen A and
Dickson, Dennis W and Graff-Radford, Neill R and Knopman,
David and Rademakers, Rosa and Lemstra, Afina W and
Pijnenburg, Yolande A L and Scheltens, Philip and Gasser,
Thomas and Chinnery, Patrick F and Hemmer, Bernhard and
Huisman, Martijn A and Troncoso, Juan and Moreno, Fermin and
Nohr, Ellen A and Sørensen, Thorkild I A and Heutink, Peter
and Sánchez-Juan, Pascual and Posthuma, Danielle and GIFT
and Clarimón, Jordi and Christensen, Kaare and
Ertekin-Taner, Nilüfer and Scholz, Sonja W and Ramirez,
Alfredo and Ruiz, Agustín and Slagboom, Eline and van der
Flier, Wiesje M and Holstege, Henne and Coppola, G. and
Karydas, A. M. and Varpetian, A. and Foroud, T. M. and
Levey, A. I. and Kukull, W. A. and Mendez, M. F. and
Ringman, J. and Chui, H. and Cotman, C. and DeCarli, C. and
Miller, B. L. and Geschwind, D. H.},
title = {{A} nonsynonymous mutation in {PLCG}2 reduces the risk of
{A}lzheimer's disease, dementia with {L}ewy bodies and
frontotemporal dementia, and increases the likelihood of
longevity.},
journal = {Acta neuropathologica},
volume = {138},
number = {2},
issn = {0001-6322},
address = {Heidelberg},
publisher = {Springer},
reportid = {DZNE-2020-07162},
pages = {237-250},
year = {2019},
abstract = {The genetic variant rs72824905-G (minor allele) in the
PLCG2 gene was previously associated with a reduced
Alzheimer's disease risk (AD). The role of PLCG2 in immune
system signaling suggests it may also protect against other
neurodegenerative diseases and possibly associates with
longevity. We studied the effect of the rs72824905-G on
seven neurodegenerative diseases and longevity, using 53,627
patients, 3,516 long-lived individuals and 149,290
study-matched controls. We replicated the association of
rs72824905-G with reduced AD risk and we found an
association with reduced risk of dementia with Lewy bodies
(DLB) and frontotemporal dementia (FTD). We did not find
evidence for an effect on Parkinson's disease (PD),
amyotrophic lateral sclerosis (ALS) and multiple sclerosis
(MS) risks, despite adequate sample sizes. Conversely, the
rs72824905-G allele was associated with increased likelihood
of longevity. By-proxy analyses in the UK Biobank supported
the associations with both dementia and longevity.
Concluding, rs72824905-G has a protective effect against
multiple neurodegenerative diseases indicating shared
aspects of disease etiology. Our findings merit studying the
PLCγ2 pathway as drug-target.},
keywords = {Alleles / Alzheimer Disease: genetics / Amyotrophic Lateral
Sclerosis: genetics / Brain: immunology / Brain: metabolism
/ Brain: pathology / Dementia: genetics / Frontotemporal
Dementia: genetics / Genetic Predisposition to Disease /
Genome-Wide Association Study / Humans / Lewy Body Disease:
genetics / Longevity: genetics / Microglia: metabolism /
Multiple Sclerosis: genetics / Mutation / Neuroimaging /
Parkinson Disease: genetics / Phospholipase C gamma:
genetics / Risk},
cin = {AG Wagner / AG Heutink 1 / U Clinical Researchers - Bonn},
ddc = {610},
cid = {I:(DE-2719)1011201 / I:(DE-2719)1210002 /
I:(DE-2719)7000001},
pnm = {344 - Clinical and Health Care Research (POF3-344) / 345 -
Population Studies and Genetics (POF3-345)},
pid = {G:(DE-HGF)POF3-344 / G:(DE-HGF)POF3-345},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:31131421},
pmc = {pmc:PMC6660501},
doi = {10.1007/s00401-019-02026-8},
url = {https://pub.dzne.de/record/140840},
}