TY - JOUR
AU - Tedeschi, Andrea
AU - Dupraz, Sebastian
AU - Curcio, Michele
AU - Laskowski, Claudia J
AU - Schaffran, Barbara
AU - Flynn, Kevin C
AU - Da Silva Santos, Telma
AU - Stern, Sina
AU - Hilton, Brett J
AU - Larson, Molly J E
AU - Gurniak, Christine B
AU - Witke, Walter
AU - Bradke, Frank
TI - ADF/Cofilin-Mediated Actin Turnover Promotes Axon Regeneration in the Adult CNS.
JO - Neuron
VL - 103
IS - 6
SN - 0896-6273
CY - New York, NY
PB - Elsevier
M1 - DZNE-2020-07840
SP - 1073-1085.e6
PY - 2019
AB - Injured axons fail to regenerate in the adult CNS, which contrasts with their vigorous growth during embryonic development. We explored the potential of re-initiating axon extension after injury by reactivating the molecular mechanisms that drive morphogenetic transformation of neurons during development. Genetic loss- and gain-of-function experiments followed by time-lapse microscopy, in vivo imaging, and whole-mount analysis show that axon regeneration is fueled by elevated actin turnover. Actin depolymerizing factor (ADF)/cofilin controls actin turnover to sustain axon regeneration after spinal cord injury through its actin-severing activity. This pinpoints ADF/cofilin as a key regulator of axon growth competence, irrespective of developmental stage. These findings reveal the central role of actin dynamics regulation in this process and elucidate a core mechanism underlying axon growth after CNS trauma. Thereby, neurons maintain the capacity to stimulate developmental programs during adult life, expanding their potential for plasticity. Thus, actin turnover is a key process for future regenerative interventions.
KW - Actins: metabolism
KW - Animals
KW - Axons: metabolism
KW - Axons: pathology
KW - Cofilin 1: genetics
KW - Cofilin 1: metabolism
KW - Cofilin 2: genetics
KW - Cofilin 2: metabolism
KW - Destrin: genetics
KW - Destrin: metabolism
KW - Growth Cones: metabolism
KW - Growth Cones: pathology
KW - Intravital Microscopy
KW - Mice
KW - Microscopy, Confocal
KW - Nerve Regeneration: genetics
KW - Neurons: metabolism
KW - Neurons: pathology
KW - Rats
KW - Spinal Cord Injuries: genetics
KW - Spinal Cord Injuries: metabolism
KW - Spinal Cord Injuries: pathology
KW - Time-Lapse Imaging
LB - PUB:(DE-HGF)16
C6 - pmid:31400829
C2 - pmc:PMC6763392
DO - DOI:10.1016/j.neuron.2019.07.007
UR - https://pub.dzne.de/record/141516
ER -