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@INBOOK{Pfaff:144653,
      author       = {Kempermann, Gerd},
      editor       = {Pfaff, Donald W.},
      title        = {{A}dult {N}eurogenesis},
      address      = {New York, NY},
      publisher    = {Springer New York},
      reportid     = {DZNE-2020-00165},
      pages        = {161-178},
      year         = {2013},
      comment      = {Neuroscience in the 21st Century / Pfaff, Donald W.
                      (Editor) ; New York, NY : Springer New York, 2013, Chapter 9
                      ; ISBN: 978-1-4614-1996-9 ; doi:10.1007/978-1-4614-1997-6},
      booktitle     = {Neuroscience in the 21st Century /
                       Pfaff, Donald W. (Editor) ; New York,
                       NY : Springer New York, 2013, Chapter 9
                       ; ISBN: 978-1-4614-1996-9 ;
                       doi:10.1007/978-1-4614-1997-6},
      abstract     = {Adult neurogenesis is the generation of new neurons in the
                      adult brain. Adult neurogenesis is an exception, as the
                      brain of mammals is non-neurogenic. Rodents and primates
                      have two neurogenic zones, the hippocampus and the olfactory
                      bulb. Lower vertebrates often have many more sites of adult
                      neurogenesis. In the peripheral nervous system, there is
                      high neurogenesis in the olfactory epithelium. Neurogenesis
                      in the olfactory bulb originates from precursor cells in the
                      wall of the lateral ventricle, the subventricular zone. New
                      interneurons in the bulb are produced. In the dentate gyrus
                      of the hippocampus, new excitatory granule cells are
                      generated that add to the mossy fiber tract into hippocampal
                      area CA3. Numerous factors are known to regulate
                      neurogenesis: it seems that adult neurogenesis responds very
                      sensitively to external stimuli. The current hypothesis is
                      that adult hippocampal neurogenesis critically contributes
                      to hippocampal function, allowing life-long adaptation
                      processes. If adult neurogenesis fails, this might
                      contribute to several important diseases, including
                      dementias, major depression and schizophrenia, as well as
                      temporal lobe epilepsy.},
      cin          = {AG Kempermann 1},
      cid          = {I:(DE-2719)1710001},
      pnm          = {342 - Disease Mechanisms and Model Systems (POF3-342)},
      pid          = {G:(DE-HGF)POF3-342},
      typ          = {PUB:(DE-HGF)7},
      doi          = {10.1007/978-1-4614-1997-6_9},
      url          = {https://pub.dzne.de/record/144653},
}