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000145019 0247_ $$2doi$$a10.1080/15476286.2019.1669879
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000145019 0247_ $$2ISSN$$a1555-8584
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000145019 037__ $$aDZNE-2020-00379
000145019 041__ $$aEnglish
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000145019 1001_ $$0P:(DE-HGF)0$$aSamir, Mohamed$$b0
000145019 245__ $$aOrgan-specific small non-coding RNA responses in domestic (Sudani) ducks experimentally infected with highly pathogenic avian influenza virus (H5N1).
000145019 260__ $$aPhiladelphia, Pa.$$bTaylor & Francis$$c2020
000145019 264_1 $$2Crossref$$3online$$bInforma UK Limited$$c2019-10-04
000145019 264_1 $$2Crossref$$3print$$bInforma UK Limited$$c2020-01-02
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000145019 520__ $$aThe duck represents an important reservoir of influenza viruses for transmission to other avian and mammalian hosts, including humans. The increased pathogenicity of the recently emerging clades of highly pathogenic avian influenza (HPAI) viruses of the H5N1 subtype in ducks features systemic viral spread and organ-to-organ variation in viral transcription and tissue damage. We previously reported that experimental infection of Sudani ducks (Cairina moschata) with an Egyptian HPAI (H5N1) virus (clade 2.2.1.2) features high viral replication and severe tissue damage in lung, but lower viral replication and only mild histological changes in brain. Little is known about the involvement of miRNA in organ-specific responses to H5N1 viruses in ducks, and involvement of the other classes of small noncoding RNA (sncRNA) has not been investigated so far. Following RNA sequencing, we have annotated the duck sncRNome and compared global expression changes of the four major sncRNA classes (miRNAs, piRNAs, snoRNAs, snRNAs) between duck lung and brain during a 120 h time course of infection with this HPAI strain. We find major organ-specific differences in miRNA, piRNA and snoRNA populations even before infection and substantial reprogramming of all sncRNA classes throughout infection, which was less pronounced in brain. Pathway prediction analysis of miRNA targets revealed enrichment of inflammation-, infection- and apoptosis-related pathways in lung, but enrichment of metabolism-related pathways (including tryptophan metabolism) in brain. Thus, organ-specific differences in sncRNA responses may contribute to differences in viral replication and organ damage in ducks infected with isolates from this emerging HPAI clade, and likely other strains.
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000145019 650_2 $$2MeSH$$aAnimals
000145019 650_2 $$2MeSH$$aChromosome Mapping
000145019 650_2 $$2MeSH$$aDucks: genetics
000145019 650_2 $$2MeSH$$aDucks: virology
000145019 650_2 $$2MeSH$$aGene Expression Profiling
000145019 650_2 $$2MeSH$$aHost-Pathogen Interactions: genetics
000145019 650_2 $$2MeSH$$aInfluenza A Virus, H5N1 Subtype: pathogenicity
000145019 650_2 $$2MeSH$$aInfluenza A Virus, H5N1 Subtype: physiology
000145019 650_2 $$2MeSH$$aInfluenza in Birds: genetics
000145019 650_2 $$2MeSH$$aInfluenza in Birds: metabolism
000145019 650_2 $$2MeSH$$aInfluenza in Birds: virology
000145019 650_2 $$2MeSH$$aMicroRNAs: genetics
000145019 650_2 $$2MeSH$$aOrgan Specificity: genetics
000145019 650_2 $$2MeSH$$aRNA, Small Untranslated: genetics
000145019 7001_ $$aVidal, Ramon O$$b1
000145019 7001_ $$0P:(DE-HGF)0$$aAbdallah, Fatma$$b2
000145019 7001_ $$aCapece, Vincenzo$$b3
000145019 7001_ $$aSeehusen, Frauke$$b4
000145019 7001_ $$aGeffers, Robert$$b5
000145019 7001_ $$aHussein, Ashraf$$b6
000145019 7001_ $$0P:(DE-HGF)0$$aAli, Ahmed A H$$b7
000145019 7001_ $$0P:(DE-2719)2810547$$aBonn, Stefan$$b8$$udzne
000145019 7001_ $$0P:(DE-HGF)0$$aPessler, Frank$$b9$$eCorresponding author
000145019 77318 $$2Crossref$$3journal-article$$a10.1080/15476286.2019.1669879$$b : Informa UK Limited, 2019-10-04$$n1$$p112-124$$tRNA Biology$$v17$$x1547-6286$$y2019
000145019 773__ $$0PERI:(DE-600)2159587-2$$a10.1080/15476286.2019.1669879$$gVol. 17, no. 1, p. 112 - 124$$n1$$p112-124$$tRNA biology$$v17$$x1547-6286$$y2020
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000145019 9141_ $$y2020
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