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@ARTICLE{TayaranianMarvian:151598,
author = {Tayaranian Marvian, Amir and Aliakbari, Farhang and
Mohammad-Beigi, Hossein and Ahmadi, Zeinab Alsadat and
Mehrpooyan, Sina and Lermyte, Frederik and Nasouti, Mahour
and Collingwood, Joanna F. and Otzen, Daniel E. and
Morshedi, Dina},
title = {{T}he status of the terminal regions of α-synuclein in
different forms of aggregates during fibrillization},
journal = {International journal of biological macromolecules},
volume = {155},
issn = {0141-8130},
address = {New York, NY [u.a.]},
publisher = {Elsevier},
reportid = {DZNE-2020-01182},
pages = {543-550},
year = {2020},
abstract = {The α-synuclein (αSN) amyloid fibrillization process is
known to be a crucial phenomenon associated with neuronal
loss in various neurodegenerative diseases, most famously
Parkinson's disease. The process involves different
aggregated species and ultimately leads to formation of
β-sheet rich fibrillar structures. Despite the essential
role of αSN aggregation in the pathoetiology of various
neurological disorders, the characteristics of various
assemblies are not fully understood. Here, we established a
fluorescence-based model for studying the end-parts of αSN
to decipher the structural aspects of aggregates during the
fibrillization. Our model proved highly sensitive to the
events at the early stage of the fibrillization process,
which are hardly detectable with routine techniques.
Combining fluorescent and PAGE analysis, we found different
oligomeric aggregates in the nucleation phase of
fibrillization with different sensitivity to SDS and
different structures based on αSN termini. Moreover, we
found that these oligomers are highly dynamic: after
reaching peak levels during fibrillization, they decline and
eventually disappear, suggesting their transformation into
other αSN aggregated species. These findings shed light on
the structural features of various αSN aggregates and their
dynamics in synucleinopathies.},
keywords = {Amyloid: chemistry / Humans / Mutant Proteins: chemistry /
Mutant Proteins: genetics / Mutant Proteins: metabolism /
Mutation / Protein Interaction Domains and Motifs / Protein
Multimerization / alpha-Synuclein: chemistry /
alpha-Synuclein: genetics / alpha-Synuclein: metabolism},
cin = {AG Höglinger 1 ; AG Höglinger 1},
ddc = {570},
cid = {I:(DE-2719)1110002},
pnm = {344 - Clinical and Health Care Research (POF3-344)},
pid = {G:(DE-HGF)POF3-344},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:32240735},
doi = {10.1016/j.ijbiomac.2020.03.238},
url = {https://pub.dzne.de/record/151598},
}