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@ARTICLE{DaSilvaSantos:153281,
author = {Da Silva Santos, Telma and Schaffran, Barbara and
Broguière, Nicolas and Meyn, Liane and Zenobi-Wong, Marcy
and Bradke, Frank},
title = {{A}xon {G}rowth of {CNS} {N}eurons in {T}hree {D}imensions
{I}s {A}moeboid and {I}ndependent of {A}dhesions},
journal = {Cell reports},
volume = {32},
number = {3},
issn = {2211-1247},
address = {[New York, NY]},
publisher = {Elsevier},
reportid = {DZNE-2020-01278},
pages = {107907},
year = {2020},
abstract = {SummaryDuring development of the central nervous system
(CNS), neurons polarize and rapidly extend their axons to
assemble neuronal circuits. The growth cone leads the axon
to its target and drives axon growth. Here, we explored the
mechanisms underlying axon growth in three dimensions. Live
in situ imaging and super-resolution microscopy combined
with pharmacological and molecular manipulations as well as
biophysical force measurements revealed that growth cones
extend CNS axons independent of pulling forces on their
substrates and without the need for adhesions in
three-dimensional (3D) environments. In 3D, microtubules
grow unrestrained from the actomyosin cytoskeleton into the
growth cone leading edge to enable rapid axon extension.
Axons extend and polarize even in adhesion-inert matrices.
Thus, CNS neurons use amoeboid mechanisms to drive axon
growth. Together with our understanding that adult CNS axons
regenerate by reactivating developmental processes, our
findings illuminate how cytoskeletal manipulations enable
axon regeneration in the adult CNS.},
keywords = {Actins: metabolism / Actomyosin: metabolism / Animals /
Axons: metabolism / Cell Adhesion / Cell Polarity / Central
Nervous System: metabolism / Collagen: metabolism /
Fibroblasts: metabolism / Growth Cones: metabolism /
Hippocampus: embryology / Mice, Inbred C57BL / Microtubules:
metabolism / Neuronal Outgrowth / Polymerization},
cin = {AG Bradke / AG Tavosanis},
ddc = {610},
cid = {I:(DE-2719)1013002 / I:(DE-2719)1013018},
pnm = {341 - Molecular Signaling (POF3-341) / 342 - Disease
Mechanisms and Model Systems (POF3-342)},
pid = {G:(DE-HGF)POF3-341 / G:(DE-HGF)POF3-342},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:32698008},
doi = {10.1016/j.celrep.2020.107907},
url = {https://pub.dzne.de/record/153281},
}