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@ARTICLE{Matschke:154197,
author = {Matschke, Jakob and Lütgehetmann, Marc and Hagel,
Christian and Sperhake, Jan P and Schröder, Ann Sophie and
Edler, Carolin and Mushumba, Herbert and Fitzek, Antonia and
Allweiss, Lena and Dandri, Maura and Dottermusch, Matthias
and Heinemann, Axel and Pfefferle, Susanne and Schwabenland,
Marius and Sumner Magruder, Daniel and Bonn, Stefan and
Prinz, Marco and Gerloff, Christian and Püschel, Klaus and
Krasemann, Susanne and Aepfelbacher, Martin and Glatzel,
Markus},
title = {{N}europathology of patients with {COVID}-19 in {G}ermany:
a post-mortem case series},
journal = {The lancet / Neurology},
volume = {19},
number = {11},
issn = {1474-4422},
address = {London},
publisher = {Lancet Publ. Group},
reportid = {DZNE-2021-00059},
pages = {919 - 929},
year = {2020},
abstract = {Background: Prominent clinical symptoms of COVID-19 include
CNS manifestations. However, it is unclear whether severe
acute respiratory syndrome coronavirus 2 (SARS-CoV-2), the
causative agent of COVID-19, gains access to the CNS and
whether it causes neuropathological changes. We investigated
the brain tissue of patients who died from COVID-19 for
glial responses, inflammatory changes, and the presence of
SARS-CoV-2 in the CNS.Methods: In this post-mortem case
series, we investigated the neuropathological features in
the brains of patients who died between March 13 and April
24, 2020, in Hamburg, Germany. Inclusion criteria comprised
a positive test for SARS-CoV-2 by quantitative RT-PCR
(qRT-PCR) and availability of adequate samples. We did a
neuropathological workup including histological staining and
immunohistochemical staining for activated astrocytes,
activated microglia, and cytotoxic T lymphocytes in the
olfactory bulb, basal ganglia, brainstem, and cerebellum.
Additionally, we investigated the presence and localisation
of SARS-CoV-2 by qRT-PCR and by immunohistochemistry in
selected patients and brain regions.Findings: 43 patients
were included in our study. Patients died in hospitals,
nursing homes, or at home, and were aged between 51 years
and 94 years (median 76 years [IQR 70-86]). We detected
fresh territorial ischaemic lesions in six $(14\%)$
patients. 37 $(86\%)$ patients had astrogliosis in all
assessed regions. Activation of microglia and infiltration
by cytotoxic T lymphocytes was most pronounced in the
brainstem and cerebellum, and meningeal cytotoxic T
lymphocyte infiltration was seen in 34 $(79\%)$ patients.
SARS-CoV-2 could be detected in the brains of 21 $(53\%)$ of
40 examined patients, with SARS-CoV-2 viral proteins found
in cranial nerves originating from the lower brainstem and
in isolated cells of the brainstem. The presence of
SARS-CoV-2 in the CNS was not associated with the severity
of neuropathological changes.Interpretation: In general,
neuropathological changes in patients with COVID-19 seem to
be mild, with pronounced neuroinflammatory changes in the
brainstem being the most common finding. There was no
evidence for CNS damage directly caused by SARS-CoV-2. The
generalisability of these findings needs to be validated in
future studies as the number of cases and availability of
clinical data were low and no age-matched and sex-matched
controls were included.Funding: German Research Foundation,
Federal State of Hamburg, EU (eRARE), German Center for
Infection Research (DZIF).},
keywords = {Aged / Aged, 80 and over / Autopsy: methods /
Betacoronavirus: isolation $\&$ purification / Brain:
pathology / Brain: virology / COVID-19 / Coronavirus
Infections: epidemiology / Coronavirus Infections: genetics
/ Coronavirus Infections: pathology / Female / Germany:
epidemiology / Humans / Male / Middle Aged / Neuropathology
/ Pandemics / Pneumonia, Viral: epidemiology / Pneumonia,
Viral: genetics / Pneumonia, Viral: pathology / SARS-CoV-2 /
Transcriptome: genetics},
cin = {AG Bonn 1},
ddc = {610},
cid = {I:(DE-2719)1410003},
pnm = {342 - Disease Mechanisms and Model Systems (POF3-342)},
pid = {G:(DE-HGF)POF3-342},
typ = {PUB:(DE-HGF)16},
pmc = {pmc:PMC7535629},
pubmed = {pmid:33031735},
doi = {10.1016/S1474-4422(20)30308-2},
url = {https://pub.dzne.de/record/154197},
}