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@ARTICLE{Enderes:154640,
author = {Enderes, Jana and Mallesh, Shilpashree and Schneider,
Reiner and Hupa, Kristof J and Lysson, Mariola and
Schneiker, Bianca and Händler, Kristian and Schlotmann,
Balthasar and Günther, Patrick and Schultze, Joachim L and
Kalff, Jörg C and Wehner, Sven},
title = {{A} {P}opulation of {R}adio-{R}esistant {M}acrophages in
the {D}eep {M}yenteric {P}lexus {C}ontributes to
{P}ostoperative {I}leus {V}ia {T}oll-{L}ike {R}eceptor 3
{S}ignaling.},
journal = {Frontiers in immunology},
volume = {11},
issn = {1664-3224},
address = {Lausanne},
publisher = {Frontiers Media},
reportid = {DZNE-2021-00288},
pages = {581111},
year = {2021},
abstract = {Postoperative ileus (POI) is triggered by an innate immune
response in the muscularis externa (ME) and is accompanied
by bacterial translocation. Bacteria can trigger an innate
immune response via toll-like receptor (TLR) activation, but
the latter's contribution to POI has been disproved for
several TLRs, including TLR2 and TLR4. Herein we
investigated the role of double-stranded RNA detection via
TLR3 and TIR-domain-containing adapter-inducing
interferon-β (TRIF) signaling pathway in POI. POI was
induced by small bowel intestinal manipulation in wt,
TRIF-/-, TLR3-/-, type I interferon receptor-/- and
interferon-β reporter mice, all on C57BL/6 background, and
POI severity was quantified by gene expression analysis,
gastrointestinal transit and leukocyte extravasation into
the ME. TRIF/TLR3 deficiency reduced postoperative ME
inflammation and prevented POI. With bone marrow
transplantation, RNA-sequencing, flow cytometry and
immunohistochemistry we revealed a distinct TLR3-expressing
radio-resistant MHCIIhiCX3CR1- IBA-1+ resident macrophage
population within the deep myenteric plexus. TLR3 deficiency
in these cells, but not in MHCIIhiCX3CR1+ macrophages,
reduced cytokine expression in POI. While this might not be
an exclusive macrophage-privileged pathway, the TLR3/TRIF
axis contributes to proinflammatory cytokine production in
MHCIIhiCX3CR1- IBA-1+ macrophages during POI. Deficiency in
TLR3/TRIF protects mice from POI. These data suggest that
TLR3 antagonism may prevent POI in humans.},
keywords = {Adaptor Proteins, Vesicular Transport: deficiency / Adaptor
Proteins, Vesicular Transport: genetics / Adaptor Proteins,
Vesicular Transport: immunology / Animals / CX3C Chemokine
Receptor 1: genetics / CX3C Chemokine Receptor 1: immunology
/ Disease Models, Animal / Female / Gene Expression / Ileus:
etiology / Ileus: immunology / Ileus: pathology / Immunity,
Innate / Macrophages: classification / Macrophages:
immunology / Macrophages: radiation effects / Mice / Mice,
Inbred C57BL / Mice, Knockout / Mice, Transgenic / Myenteric
Plexus: immunology / Postoperative Complications: etiology /
Postoperative Complications: immunology / Postoperative
Complications: pathology / Radiation Tolerance: immunology /
Receptor, Interferon alpha-beta: deficiency / Receptor,
Interferon alpha-beta: genetics / Receptor, Interferon
alpha-beta: immunology / Signal Transduction: immunology /
Toll-Like Receptor 3: deficiency / Toll-Like Receptor 3:
genetics / Toll-Like Receptor 3: immunology /
Transplantation Chimera: immunology / TLR3 (Other) / TRIF
(Other) / innate immune response (Other) / macrophages
(Other) / postoperative ileus (Other)},
cin = {PRECISE},
ddc = {610},
cid = {I:(DE-2719)1013031},
pnm = {341 - Molecular Signaling (POF3-341) / 352 - Disease
Mechanisms (POF4-352) / 354 - Disease Prevention and Healthy
Aging (POF4-354)},
pid = {G:(DE-HGF)POF3-341 / G:(DE-HGF)POF4-352 /
G:(DE-HGF)POF4-354},
experiment = {EXP:(DE-2719)PRECISE-20190321},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:33519804},
pmc = {pmc:PMC7838642},
doi = {10.3389/fimmu.2020.581111},
url = {https://pub.dzne.de/record/154640},
}