TY - JOUR
AU - Colombo, Alessio Vittorio
AU - Dinkel, Lina
AU - Müller, Stephan A
AU - Sebastian Monasor, Laura
AU - Schifferer, Martina
AU - Cantuti-Castelvetri, Ludovico
AU - König, Jasmin
AU - Vidatic, Lea
AU - Bremova-Ertl, Tatiana
AU - Lieberman, Andrew P.
AU - Hecimovic, Silva
AU - Simons, Mikael
AU - Lichtenthaler, Stefan F.
AU - Strupp, Michael
AU - Schneider, Susanne A.
AU - Tahirovic, Sabina
TI - Loss of NPC1 enhances phagocytic uptake and impairs lipid trafficking in microglia
JO - Nature Communications
VL - 12
IS - 1
SN - 2041-1723
CY - [London]
PB - Nature Publishing Group UK
M1 - DZNE-2021-00409
SP - 1158
PY - 2021
AB - Niemann-Pick type C disease is a rare neurodegenerative disorder mainly caused by mutations in NPC1, resulting in abnormal late endosomal/lysosomal lipid storage. Although microgliosis is a prominent pathological feature, direct consequences of NPC1 loss on microglial function remain not fully characterized. We discovered pathological proteomic signatures and phenotypes in NPC1-deficient murine models and demonstrate a cell autonomous function of NPC1 in microglia. Loss of NPC1 triggers enhanced phagocytic uptake and impaired myelin turnover in microglia that precede neuronal death. Npc1-/- microglia feature a striking accumulation of multivesicular bodies and impaired trafficking of lipids to lysosomes while lysosomal degradation function remains preserved. Molecular and functional defects were also detected in blood-derived macrophages of NPC patients that provide a potential tool for monitoring disease. Our study underscores an essential cell autonomous role for NPC1 in immune cells and implies microglial therapeutic potential.
KW - Animals
KW - Blotting, Western
KW - Cells, Cultured
KW - Cholesterol: metabolism
KW - Female
KW - Humans
KW - Intracellular Signaling Peptides and Proteins: genetics
KW - Intracellular Signaling Peptides and Proteins: metabolism
KW - Male
KW - Mass Spectrometry
KW - Mice
KW - Mice, Inbred C57BL
KW - Mice, Knockout
KW - Microglia: metabolism
KW - Myelin Sheath: metabolism
KW - Niemann-Pick C1 Protein
KW - Niemann-Pick Disease, Type C: genetics
KW - Niemann-Pick Disease, Type C: metabolism
KW - Phagocytosis: genetics
KW - Phagocytosis: physiology
KW - Proteomics: methods
LB - PUB:(DE-HGF)16
C6 - pmid:33627648
C6 - 33627648
C2 - pmc:PMC7904859
DO - DOI:10.1038/s41467-021-21428-5
UR - https://pub.dzne.de/record/154831
ER -