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@ARTICLE{Lo:155556,
      author       = {Lo, Eng H and Albers, Gregory W and Dichgans, Martin and
                      Donnan, Geoffrey and Esposito, Elga and Foster, Russell and
                      Howells, David W and Huang, Yi-Ge and Ji, Xunming and
                      Klerman, Elizabeth B and Lee, Sarah and Li, Wenlu and
                      Liebeskind, David S and Lizasoain, Ignacio and Mandeville,
                      Emiri T and Moro, Maria A and Ning, MingMing and Ray, David
                      and Sakadžić, Sava and Saver, Jeffrey L and Scheer, Frank
                      A J L and Selim, Magdy and Tiedt, Steffen and Zhang, Fang
                      and Buchan, Alastair M},
      title        = {{C}ircadian {B}iology and {S}troke.},
      journal      = {Stroke},
      volume       = {52},
      number       = {6},
      issn         = {1524-4628},
      address      = {Philadelphia, Pa.},
      publisher    = {Lippincott Williams $\&$ Wilkins},
      reportid     = {DZNE-2021-00734},
      pages        = {2180 - 2190},
      year         = {2021},
      abstract     = {Circadian biology modulates almost all aspects of mammalian
                      physiology, disease, and response to therapies. Emerging
                      data suggest that circadian biology may significantly affect
                      the mechanisms of susceptibility, injury, recovery, and the
                      response to therapy in stroke. In this review/perspective,
                      we survey the accumulating literature and attempt to connect
                      molecular, cellular, and physiological pathways in circadian
                      biology to clinical consequences in stroke. Accounting for
                      the complex and multifactorial effects of circadian rhythm
                      may improve translational opportunities for stroke
                      diagnostics and therapeutics.},
      subtyp        = {Review Article},
      keywords     = {Animals / Circadian Rhythm: physiology / Clinical Trials as
                      Topic: methods / Humans / Inflammation Mediators: physiology
                      / Neurovascular Coupling: physiology / Stroke: diagnosis /
                      Stroke: physiopathology / Stroke: therapy / biomarkers
                      (Other) / circadian rhythm (Other) / immune system (Other) /
                      ischemia (Other) / neuroprotection (Other) / sleep (Other)},
      cin          = {AG Höglinger 2},
      ddc          = {610},
      cid          = {I:(DE-2719)1111015},
      pnm          = {353 - Clinical and Health Care Research (POF4-353)},
      pid          = {G:(DE-HGF)POF4-353},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:33940951},
      doi          = {10.1161/STROKEAHA.120.031742},
      url          = {https://pub.dzne.de/record/155556},
}