TY - JOUR
AU - Stenton, Sarah L
AU - Sheremet, Natalia L
AU - Catarino, Claudia B
AU - Andreeva, Natalia A
AU - Assouline, Zahra
AU - Barboni, Piero
AU - Barel, Ortal
AU - Berutti, Riccardo
AU - Bychkov, Igor
AU - Caporali, Leonardo
AU - Capristo, Mariantonietta
AU - Carbonelli, Michele
AU - Cascavilla, Maria L
AU - Charbel Issa, Peter
AU - Freisinger, Peter
AU - Gerber, Sylvie
AU - Ghezzi, Daniele
AU - Graf, Elisabeth
AU - Heidler, Juliana
AU - Hempel, Maja
AU - Heon, Elise
AU - Itkis, Yulya S
AU - Javasky, Elisheva
AU - Kaplan, Josseline
AU - Kopajtich, Robert
AU - Kornblum, Cornelia
AU - Kovacs-Nagy, Reka
AU - Krylova, Tatiana D
AU - Kunz, Wolfram S
AU - La Morgia, Chiara
AU - Lamperti, Costanza
AU - Ludwig, Christina
AU - Malacarne, Pedro F
AU - Maresca, Alessandra
AU - Mayr, Johannes A
AU - Meisterknecht, Jana
AU - Nevinitsyna, Tatiana A
AU - Palombo, Flavia
AU - Pode-Shakked, Ben
AU - Shmelkova, Maria S
AU - Strom, Tim M
AU - Tagliavini, Francesca
AU - Tzadok, Michal
AU - van der Ven, Amelie T
AU - Vignal-Clermont, Catherine
AU - Wagner, Matias
AU - Zakharova, Ekaterina Y
AU - Zhorzholadze, Nino V
AU - Rozet, Jean-Michel
AU - Carelli, Valerio
AU - Tsygankova, Polina G
AU - Klopstock, Thomas
AU - Wittig, Ilka
AU - Prokisch, Holger
TI - Impaired complex I repair causes recessive Leber's hereditary optic neuropathy.
JO - The journal of clinical investigation
VL - 131
IS - 6
SN - 1558-8238
CY - Ann Arbor, Mich.
PB - ASCJ
M1 - DZNE-2021-01184
SP - e138267
PY - 2021
AB - Leber's hereditary optic neuropathy (LHON) is the most frequent mitochondrial disease and was the first to be genetically defined by a point mutation in mitochondrial DNA (mtDNA). A molecular diagnosis is achieved in up to 95
KW - Adolescent
KW - Adult
KW - Cell Line
KW - Child, Preschool
KW - Electron Transport Complex I: chemistry
KW - Electron Transport Complex I: metabolism
KW - Female
KW - Gene Knockout Techniques
KW - Genes, Recessive
KW - HSP40 Heat-Shock Proteins: deficiency
KW - HSP40 Heat-Shock Proteins: genetics
KW - HSP40 Heat-Shock Proteins: metabolism
KW - Homozygote
KW - Humans
KW - Male
KW - Middle Aged
KW - Mutation
KW - Optic Atrophy, Hereditary, Leber: genetics
KW - Optic Atrophy, Hereditary, Leber: metabolism
KW - Pedigree
KW - Penetrance
KW - Phenotype
KW - Protein Subunits
KW - Reactive Oxygen Species: metabolism
KW - Young Adult
KW - Genetic diseases (Other)
KW - Genetics (Other)
KW - Neuroscience (Other)
LB - PUB:(DE-HGF)16
C6 - pmid:33465056
C2 - pmc:PMC7954600
DO - DOI:10.1172/JCI138267
UR - https://pub.dzne.de/record/157727
ER -