% IMPORTANT: The following is UTF-8 encoded. This means that in the presence
% of non-ASCII characters, it will not work with BibTeX 0.99 or older.
% Instead, you should use an up-to-date BibTeX implementation like “bibtex8” or
% “biber”.
@ARTICLE{Lu:162239,
author = {Lu, Ran and Aziz, N Ahmad and Reuter, Martin and Stöcker,
Tony and Breteler, Monique M B},
title = {{E}valuation of the {N}euroanatomical {B}asis of
{O}lfactory {D}ysfunction in the {G}eneral {P}opulation},
journal = {JAMA otolaryngology - head $\&$ neck surgery},
volume = {147},
number = {10},
issn = {2168-6181},
address = {Chicago, Ill.},
publisher = {American Medical Association},
reportid = {DZNE-2021-01293},
pages = {855 - 863},
year = {2021},
abstract = {Olfactory dysfunction is a prodromal manifestation of many
neurodegenerative disorders, including Alzheimer and
Parkinson disease. However, its neuroanatomical basis is
largely unknown.To assess the association between olfactory
brain structures and olfactory function in adults 30 years
or older and to examine the extent to which olfactory bulb
volume (OBV) mediates the association between central
olfactory structures and olfactory function.This
cross-sectional study analyzed baseline data from the first
639 participants with brain magnetic resonance imaging (MRI)
in the Rhineland Study, an ongoing population-based cohort
study in Bonn, Germany. Participants were enrolled between
March 7, 2016, and October 31, 2017, and underwent brain MRI
and olfactory assessment. Data were analyzed from March 1,
2018, to June 30, 2021.Volumetric measures were derived from
3-T MRI T1-weighted brain scans, and OBV was manually
segmented on T2-weighted images. The mean volumetric brain
measures from the right and left sides were calculated,
adjusted by head size, and normalized to all
participants.Performance on the 12-item smell identification
test (SIT-12) was used as a proxy for olfactory function.A
total of 541 participants with complete data on MRI-derived
measures and SIT-12 scores were included. This population
had a mean (SD) age of 53.6 (13.1) years and comprised 306
women $(56.6\%).$ Increasing age (difference in SIT-12
score, -0.04; $95\%$ CI, -0.05 to -0.03), male sex (-0.26;
$95\%$ CI, -0.54 to 0.02), and nasal congestion (-0.28;
$95\%$ CI, -0.66 to 0.09) were associated with worse
olfactory function (SIT-12 scores). Conversely, larger OBV
was associated with better olfactory function (difference in
SIT-12 score, 0.46; $95\%$ CI, 0.29-0.64). Larger volumes of
amygdala (difference in OBV, 0.12; $95\%$ CI, 0.01-0.24),
hippocampus (0.16; $95\%$ CI, 0.04-0.28), insular cortex
(0.12; $95\%$ CI, 0.01-0.24), and medial orbitofrontal
cortex (0.10; $95\%$ CI, 0.00-0.20) were associated with
larger OBV. Larger volumes of amygdala (volume × age
interaction effect, 0.17; $95\%$ CI, 0.03-0.30),
parahippocampal cortex (0.17; $95\%$ CI, 0.03-0.31), and
hippocampus (0.21; $95\%$ CI, 0.08-0.35) were associated
with better olfactory function only in older age groups. The
age-modified association between volumes of central
olfactory structures and olfactory function was largely
mediated through OBV.This cross-sectional study found that
olfactory bulb volume was independently associated with odor
identification function and was a robust mediator of the
age-dependent association between volumes of central
olfactory structures and olfactory function. Thus,
neurodegeneration-associated olfactory dysfunction may
primarily originate from the pathology of peripheral
olfactory structures, suggesting that OBV may serve as a
preclinical marker for the identification of individuals who
are at an increased risk of neurodegenerative diseases.},
keywords = {Adult / Aged / Amygdala: diagnostic imaging / Cerebral
Cortex: diagnostic imaging / Cross-Sectional Studies /
Female / Germany / Hippocampus: diagnostic imaging / Humans
/ Magnetic Resonance Imaging / Male / Middle Aged /
Olfaction Disorders: diagnostic imaging / Olfaction
Disorders: physiopathology / Olfactory Bulb: diagnostic
imaging / Organ Size / Prefrontal Cortex: diagnostic
imaging},
cin = {AG Aziz / AG Reuter / AG Stöcker / AG Breteler},
ddc = {610},
cid = {I:(DE-2719)5000071 / I:(DE-2719)1040310 /
I:(DE-2719)1013026 / I:(DE-2719)1012001},
pnm = {354 - Disease Prevention and Healthy Aging (POF4-354)},
pid = {G:(DE-HGF)POF4-354},
experiment = {EXP:(DE-2719)Rhineland Study-20190321},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:34436517},
pmc = {pmc:PMC8391780},
doi = {10.1001/jamaoto.2021.2026},
url = {https://pub.dzne.de/record/162239},
}