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@INBOOK{Ermer:162762,
      author       = {Ermer, Veronika and Nicotera, Pierluigi and Stainbrook,
                      Sarah C.},
      title        = {{C}ell {D}eath | {C}ell {D}eath by {A}poptosis and
                      {N}ecrosis},
      volume       = {5},
      publisher    = {Elsevier},
      reportid     = {DZNE-2021-01418},
      pages        = {38-41},
      year         = {2021},
      comment      = {Encyclopedia of Biological Chemistry III / Ermer, Veronika
                      ; : Elsevier, 2021, ; ISBN: 9780128220405 ;
                      doi:10.1016/B978-0-12-819460-7.00270-X},
      booktitle     = {Encyclopedia of Biological Chemistry
                       III / Ermer, Veronika ; : Elsevier,
                       2021, ; ISBN: 9780128220405 ;
                       doi:10.1016/B978-0-12-819460-7.00270-X},
      abstract     = {Various genetically encoded programs involved in the
                      signaling, initiation, and execution of cell death decide
                      cells׳ fate during development and adult life. These
                      programs can execute physiological cell death during
                      development or tissue turnover, and are also involved in the
                      inappropriate elimination of cells under pathological
                      conditions. Because balanced cell turnover is essential for
                      life, defects in cell elimination can also result in
                      disease, the foremost example being cancer. In many
                      circumstances, both physiological cell death and cell death
                      in pathological settings have similar morphological and
                      biochemical characteristics. Perhaps the best-characterized
                      biochemical and morphological changes during a cell death
                      program are those defined as apoptosis. Apoptosis is
                      characterized by condensation and fragmentation of the
                      nucleus with shrinkage of the cytoplasm and exposure of
                      surface molecules that facilitate recognition of the dying
                      cells by phagocytes. However, other types of cell death are
                      present and are strictly regulated in vivo, including cell
                      lysis/necrosis or autophagy. Imbalance in cellular calcium
                      regulation has been involved in both apoptotic and
                      nonapoptotic cell death. Calcium can be a signal for cell
                      death or simply a downstream consequence of the activation
                      of the death machinery.},
      cin          = {AG Nicotera / Scientific board / AG Bano},
      cid          = {I:(DE-2719)5000018 / I:(DE-2719)1030000 /
                      I:(DE-2719)1013003},
      pnm          = {351 - Brain Function (POF4-351)},
      pid          = {G:(DE-HGF)POF4-351},
      typ          = {PUB:(DE-HGF)7},
      doi          = {10.1016/B978-0-12-819460-7.00270-X},
      url          = {https://pub.dzne.de/record/162762},
}