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@ARTICLE{Leiter:163270,
author = {Leiter, Odette and Zhuo, Zhan and Rust, Ruslan and
Wasielewska, Joanna M and Grönnert, Lisa and Kowal, Susann
and Overall, Rupert W and Adusumilli, Vijay S and Blackmore,
Daniel G and Southon, Adam and Ganio, Katherine and
McDevitt, Christopher A and Rund, Nicole and Brici, David
and Mudiyan, Imesh Aththanayake and Sykes, Alexander M and
Rünker, Annette E and Zocher, Sara and Ayton, Scott and
Bush, Ashley I and Bartlett, Perry F and Hou, Sheng-Tao and
Kempermann, Gerd and Walker, Tara L},
title = {{S}elenium mediates exercise-induced adult neurogenesis and
reverses learning deficits induced by hippocampal injury and
aging.},
journal = {Cell metabolism},
volume = {34},
number = {3},
issn = {1550-4131},
address = {Cambridge, Mass.},
publisher = {Cell Press},
reportid = {DZNE-2022-00050},
pages = {408 - 423.e8},
year = {2022},
note = {(CC BY-NC-ND)},
abstract = {Although the neurogenesis-enhancing effects of exercise
have been extensively studied, the molecular mechanisms
underlying this response remain unclear. Here, we propose
that this is mediated by the exercise-induced systemic
release of the antioxidant selenium transport protein,
selenoprotein P (SEPP1). Using knockout mouse models, we
confirmed that SEPP1 and its receptor low-density
lipoprotein receptor-related protein 8 (LRP8) are required
for the exercise-induced increase in adult hippocampal
neurogenesis. In vivo selenium infusion increased
hippocampal neural precursor cell (NPC) proliferation and
adult neurogenesis. Mimicking the effect of exercise through
dietary selenium supplementation restored neurogenesis and
reversed the cognitive decline associated with aging and
hippocampal injury, suggesting potential therapeutic
relevance. These results provide a molecular mechanism
linking exercise-induced changes in the systemic environment
to the activation of quiescent hippocampal NPCs and their
subsequent recruitment into the neurogenic trajectory.},
keywords = {Aging / Animals / Cell Proliferation / Hippocampus / Mice /
Neural Stem Cells: metabolism / Neurogenesis: physiology /
Selenium: metabolism / Selenium: pharmacology / adult
neurogenesis (Other) / aging (Other) / dentate gyrus (Other)
/ endothelin-1 (Other) / exercise (Other) / hippocampal
lesion (Other) / hippocampus (Other) / neural precursor cell
(Other) / neural stem cell (Other) / selenium (Other)},
cin = {Dresden common / AG Kempermann / AG Toda},
ddc = {570},
cid = {I:(DE-2719)6000013 / I:(DE-2719)1710001 /
I:(DE-2719)1710014},
pnm = {352 - Disease Mechanisms (POF4-352)},
pid = {G:(DE-HGF)POF4-352},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:35120590},
doi = {10.1016/j.cmet.2022.01.005},
url = {https://pub.dzne.de/record/163270},
}