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@ARTICLE{Pagano:165174,
author = {Pagano, Gennaro and Taylor, Kirsten I and Anzures-Cabrera,
Judith and Marchesi, Maddalena and Simuni, Tanya and Marek,
Kenneth and Postuma, Ronald B and Pavese, Nicola and
Stocchi, Fabrizio and Azulay, Jean-Philippe and Mollenhauer,
Brit and López-Manzanares, Lydia and Russell, David S and
Boyd, James T and Nicholas, Anthony P and Luquin, María R
and Hauser, Robert A and Gasser, Thomas and Poewe, Werner
and Ricci, Benedicte and Boulay, Anne and Vogt, Annamarie
and Boess, Frank G and Dukart, Juergen and D'Urso, Giulia
and Finch, Rebecca and Zanigni, Stefano and Monnet,
Annabelle and Pross, Nathalie and Hahn, Andrea and Svoboda,
Hanno and Britschgi, Markus and Lipsmeier, Florian and
Volkova-Volkmar, Ekaterina and Lindemann, Michael and
Dziadek, Sebastian and Holiga, Štefan and Rukina, Daria and
Kustermann, Thomas and Kerchner, Geoffrey A and Fontoura,
Paulo and Umbricht, Daniel and Doody, Rachelle and
Nikolcheva, Tania and Bonni, Azad},
collaboration = {Investigators, PASADENA and Group, Prasinezumab Study},
title = {{T}rial of {P}rasinezumab in {E}arly-{S}tage {P}arkinson's
{D}isease.},
journal = {The New England journal of medicine},
volume = {387},
number = {5},
issn = {0028-4793},
address = {Waltham, Mass.},
publisher = {MMS},
reportid = {DZNE-2022-01476},
pages = {421 - 432},
year = {2022},
abstract = {Aggregated α-synuclein plays an important role in the
pathogenesis of Parkinson's disease. The monoclonal antibody
prasinezumab, directed at aggregated α-synuclein, is being
studied for its effect on Parkinson's disease.In this phase
2 trial, we randomly assigned participants with early-stage
Parkinson's disease in a 1:1:1 ratio to receive intravenous
placebo or prasinezumab at a dose of 1500 mg or 4500 mg
every 4 weeks for 52 weeks. The primary end point was the
change from baseline to week 52 in the sum of scores on
parts I, II, and III of the Movement Disorder
Society-sponsored revision of the Unified Parkinson's
Disease Rating Scale (MDS-UPDRS; range, 0 to 236, with
higher scores indicating greater impairment). Secondary end
points included the dopamine transporter levels in the
putamen of the hemisphere ipsilateral to the clinically more
affected side of the body, as measured by 123I-ioflupane
single-photon-emission computed tomography (SPECT).A total
of 316 participants were enrolled; 105 were assigned to
receive placebo, 105 to receive 1500 mg of prasinezumab, and
106 to receive 4500 mg of prasinezumab. The baseline mean
MDS-UPDRS scores were 32.0 in the placebo group, 31.5 in the
1500-mg group, and 30.8 in the 4500-mg group, and mean
(±SE) changes from baseline to 52 weeks were 9.4±1.2 in
the placebo group, 7.4±1.2 in the 1500-mg group (difference
vs. placebo, -2.0; $80\%$ confidence interval [CI], -4.2 to
0.2; P = 0.24), and 8.8±1.2 in the 4500-mg group
(difference vs. placebo, -0.6; $80\%$ CI, -2.8 to 1.6; P =
0.72). There was no substantial difference between the
active-treatment groups and the placebo group in dopamine
transporter levels on SPECT. The results for most clinical
secondary end points were similar in the active-treatment
groups and the placebo group. Serious adverse events
occurred in $6.7\%$ of the participants in the 1500-mg group
and in $7.5\%$ of those in the 4500-mg group; infusion
reactions occurred in $19.0\%$ and $34.0\%,$
respectively.Prasinezumab therapy had no meaningful effect
on global or imaging measures of Parkinson's disease
progression as compared with placebo and was associated with
infusion reactions. (Funded by F. Hoffmann-La Roche and
Prothena Biosciences; PASADENA ClinicalTrials.gov number,
NCT03100149.).},
keywords = {Antibodies, Monoclonal, Humanized: therapeutic use /
Antiparkinson Agents: therapeutic use / Dopamine Plasma
Membrane Transport Proteins: therapeutic use / Double-Blind
Method / Humans / Parkinson Disease: drug therapy /
Treatment Outcome / alpha-Synuclein: antagonists $\&$
inhibitors / Antibodies, Monoclonal, Humanized (NLM
Chemicals) / Antiparkinson Agents (NLM Chemicals) / Dopamine
Plasma Membrane Transport Proteins (NLM Chemicals) /
alpha-Synuclein (NLM Chemicals)},
cin = {AG Gasser},
ddc = {610},
cid = {I:(DE-2719)1210000},
pnm = {353 - Clinical and Health Care Research (POF4-353)},
pid = {G:(DE-HGF)POF4-353},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:35921451},
doi = {10.1056/NEJMoa2202867},
url = {https://pub.dzne.de/record/165174},
}