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@ARTICLE{Griffin:249930,
author = {Griffin, Jarred and Hingorani Jai Prakash, Sonia and
Bockemühl, Till and Benner, Jessica M and Schaffran,
Barbara and Moreno-Manzano, Victoria and Büschges, Ansgar
and Bradke, Frank},
title = {{R}ehabilitation enhances epothilone-induced locomotor
recovery after spinal cord injury.},
journal = {Brain communications},
volume = {5},
number = {1},
issn = {2632-1297},
address = {[Großbritannien]},
publisher = {Guarantors of Brain},
reportid = {DZNE-2023-00245},
pages = {fcad005},
year = {2023},
abstract = {Microtubule stabilization through epothilones is a
promising preclinical therapy for functional recovery
following spinal cord injury that stimulates axon
regeneration, reduces growth-inhibitory molecule deposition
and promotes functional improvements. Rehabilitation therapy
is the only clinically validated approach to promote
functional improvements following spinal cord injury.
However, whether microtubule stabilization can augment the
beneficial effects of rehabilitation therapy or act in
concert with it to further promote repair remains unknown.
Here, we investigated the pharmacokinetic, histological and
functional efficacies of epothilone D, epothilone B and
ixabepilone alone or in combination with rehabilitation
following a moderate contusive spinal cord injury.
Pharmacokinetic analysis revealed that ixabepilone only
weakly crossed the blood-brain barrier and was subsequently
excluded from further investigations. In contrast,
epothilones B and D rapidly distributed to CNS compartments
displaying similar profiles after either subcutaneous or
intraperitoneal injections. Following injury and
subcutaneous administration of epothilone B or D, rats were
subjected to 7 weeks of sequential bipedal and quadrupedal
training. For all outcome measures, epothilone B was
efficacious compared with epothilone D. Specifically,
epothilone B decreased fibrotic scaring which was associated
with a retention of fibronectin localized to perivascular
cells in sections distal to the lesion. This corresponded to
a decreased number of cells present within the intralesional
space, resulting in less axons within the lesion. Instead,
epothilone B increased serotonergic fibre regeneration and
vesicular glutamate transporter 1 expression caudal to the
lesion, which was not affected by rehabilitation.
Multiparametric behavioural analyses consisting of
open-field locomotor scoring, horizontal ladder, catwalk
gait analysis and hindlimb kinematics revealed that
rehabilitation and epothilone B both improved several
aspects of locomotion. Specifically, rehabilitation improved
open-field locomotor and ladder scores, as well as improving
the gait parameters of limb coupling, limb support, stride
length and limb speed; epothilone B improved these same gait
parameters but also hindlimb kinematic profiles. Functional
improvements by epothilone B and rehabilitation acted
complementarily on gait parameters leading to an enhanced
recovery in the combination group. As a result, principal
component analysis of gait showed the greatest improvement
in the epothilone B plus rehabilitation group. Thus, these
results support the combination of epothilone B with
rehabilitation in a clinical setting.},
keywords = {axon regeneration (Other) / epothilone (Other) /
neuroplasticity (Other) / rehabilitation (Other) / spinal
cord injury (Other)},
cin = {AG Bradke},
ddc = {610},
cid = {I:(DE-2719)1013002},
pnm = {351 - Brain Function (POF4-351)},
pid = {G:(DE-HGF)POF4-351},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:36744011},
pmc = {pmc:PMC9893225},
doi = {10.1093/braincomms/fcad005},
url = {https://pub.dzne.de/record/249930},
}