TY - JOUR
AU - Meyer, Thomas
AU - Schumann, Peggy
AU - Weydt, Patrick
AU - Petri, Susanne
AU - Koc, Yasemin
AU - Spittel, Susanne
AU - Bernsen, Sarah
AU - Günther, René
AU - Weishaupt, Jochen H
AU - Dreger, Marie
AU - Kolzarek, Felix
AU - Kettemann, Dagmar
AU - Norden, Jenny
AU - Boentert, Matthias
AU - Vidovic, Maximilian
AU - Meisel, Christian
AU - Münch, Christoph
AU - Maier, André
AU - Körtvélyessy, Péter
TI - Neurofilament light-chain response during therapy with antisense oligonucleotide tofersen in SOD1-related ALS: Treatment experience in clinical practice.
JO - Muscle & nerve
VL - 67
IS - 6
SN - 0148-639X
CY - New York, NY [u.a.]
PB - Wiley
M1 - DZNE-2023-00466
SP - 515 - 521
PY - 2023
AB - In amyotrophic lateral sclerosis (ALS) caused by superoxide dismutase 1 (SOD1) gene mutations (SOD1-ALS), the antisense oligonucleotide tofersen had been investigated in a phase III study (VALOR) and subsequently introduced in an expanded access program. In this study we assess neurofilament light chain (NfL) before and during tofersen treatment.In six SOD1-ALS patients treated with tofersen at three specialized ALS centers in Germany, NfL in cerebrospinal fluid (CSF-NfL) and/or serum (sNfL) were investigated using the ALS Functional Rating Scale Revised (ALSFRS-R) and ALS progression rate (ALS-PR), defined by monthly decline of ALSFRS-R.Three of the six SOD1-ALS patients reported a negative family history. Three patients harbored a homozygous c.272A > C, p.(Asp91Ala) mutation. These and two other patients showed slower progressing ALS (defined by ALS-PR <0.9), whereas one patient demonstrated rapidly progressing ALS (ALS-PR = 2.66). Mean treatment duration was 6.5 (range 5 to 8) months. In all patients, NfL decreased (mean CSF-NfL: -66
KW - Humans
KW - Amyotrophic Lateral Sclerosis: drug therapy
KW - Amyotrophic Lateral Sclerosis: genetics
KW - Oligonucleotides, Antisense: therapeutic use
KW - Superoxide Dismutase-1: genetics
KW - Intermediate Filaments
KW - Biomarkers
KW - Neurofilament Proteins
KW - tofersen (NLM Chemicals)
KW - amyotrophic lateral sclerosis (Other)
KW - neurofilament light chain (Other)
KW - tofersen (Other)
KW - Oligonucleotides, Antisense (NLM Chemicals)
KW - Superoxide Dismutase-1 (NLM Chemicals)
KW - Biomarkers (NLM Chemicals)
KW - Neurofilament Proteins (NLM Chemicals)
KW - SOD1 protein, human (NLM Chemicals)
LB - PUB:(DE-HGF)16
C6 - pmid:36928619
DO - DOI:10.1002/mus.27818
UR - https://pub.dzne.de/record/257590
ER -