000257894 001__ 257894
000257894 005__ 20240624140128.0
000257894 0247_ $$2doi$$a10.1038/s41418-023-01153-w
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000257894 0247_ $$2ISSN$$a1476-5403
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000257894 037__ $$aDZNE-2023-00522
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000257894 1001_ $$00000-0002-5918-1841$$aVitale, Ilio$$b0
000257894 245__ $$aApoptotic cell death in disease-Current understanding of the NCCD 2023.
000257894 260__ $$aLondon$$bMacmillan$$c2023
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000257894 520__ $$aApoptosis is a form of regulated cell death (RCD) that involves proteases of the caspase family. Pharmacological and genetic strategies that experimentally inhibit or delay apoptosis in mammalian systems have elucidated the key contribution of this process not only to (post-)embryonic development and adult tissue homeostasis, but also to the etiology of multiple human disorders. Consistent with this notion, while defects in the molecular machinery for apoptotic cell death impair organismal development and promote oncogenesis, the unwarranted activation of apoptosis promotes cell loss and tissue damage in the context of various neurological, cardiovascular, renal, hepatic, infectious, neoplastic and inflammatory conditions. Here, the Nomenclature Committee on Cell Death (NCCD) gathered to critically summarize an abundant pre-clinical literature mechanistically linking the core apoptotic apparatus to organismal homeostasis in the context of disease.
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000257894 650_7 $$0EC 3.4.22.-$$2NLM Chemicals$$aCaspases
000257894 650_2 $$2MeSH$$aAnimals
000257894 650_2 $$2MeSH$$aHumans
000257894 650_2 $$2MeSH$$aApoptosis: genetics
000257894 650_2 $$2MeSH$$aCell Death
000257894 650_2 $$2MeSH$$aCaspases: genetics
000257894 650_2 $$2MeSH$$aCaspases: metabolism
000257894 650_2 $$2MeSH$$aCarcinogenesis
000257894 650_2 $$2MeSH$$aMammals: metabolism
000257894 7001_ $$aPietrocola, Federico$$b1
000257894 7001_ $$aGuilbaud, Emma$$b2
000257894 7001_ $$aAaronson, Stuart A$$b3
000257894 7001_ $$aAbrams, John M$$b4
000257894 7001_ $$aAdam, Dieter$$b5
000257894 7001_ $$aAgostini, Massimiliano$$b6
000257894 7001_ $$aAgostinis, Patrizia$$b7
000257894 7001_ $$aAlnemri, Emad S$$b8
000257894 7001_ $$aAltucci, Lucia$$b9
000257894 7001_ $$aAmelio, Ivano$$b10
000257894 7001_ $$aAndrews, David W$$b11
000257894 7001_ $$aAqeilan, Rami I$$b12
000257894 7001_ $$aArama, Eli$$b13
000257894 7001_ $$aBaehrecke, Eric H$$b14
000257894 7001_ $$aBalachandran, Siddharth$$b15
000257894 7001_ $$0P:(DE-2719)2158358$$aBano, Daniele$$b16$$udzne
000257894 7001_ $$aBarlev, Nickolai A$$b17
000257894 7001_ $$aBartek, Jiri$$b18
000257894 7001_ $$aBazan, Nicolas G$$b19
000257894 7001_ $$aBecker, Christoph$$b20
000257894 7001_ $$aBernassola, Francesca$$b21
000257894 7001_ $$aBertrand, Mathieu J M$$b22
000257894 7001_ $$aBianchi, Marco E$$b23
000257894 7001_ $$aBlagosklonny, Mikhail V$$b24
000257894 7001_ $$aBlander, J Magarian$$b25
000257894 7001_ $$aBlandino, Giovanni$$b26
000257894 7001_ $$aBlomgren, Klas$$b27
000257894 7001_ $$aBorner, Christoph$$b28
000257894 7001_ $$aBortner, Carl D$$b29
000257894 7001_ $$aBove, Pierluigi$$b30
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000257894 7001_ $$aDamgaard, Rune Busk$$b35
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000257894 7001_ $$aFimia, Gian Maria$$b72
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000257894 7001_ $$aGarg, Abhishek D$$b76
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000257894 7001_ $$aHe, Sudan$$b88
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000257894 7001_ $$aYamazaki, Takahiro$$b217
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000257894 7001_ $$aZakeri, Zahra$$b219
000257894 7001_ $$aZawacka-Pankau, Joanna E$$b220
000257894 7001_ $$aZhang, Lin$$b221
000257894 7001_ $$aZhang, Haibing$$b222
000257894 7001_ $$aZhivotovsky, Boris$$b223
000257894 7001_ $$aZhou, Wenzhao$$b224
000257894 7001_ $$00000-0003-2919-1296$$aPiacentini, Mauro$$b225
000257894 7001_ $$00000-0002-9334-4405$$aKroemer, Guido$$b226
000257894 7001_ $$00000-0003-2257-8500$$aGalluzzi, Lorenzo$$b227
000257894 773__ $$0PERI:(DE-600)1496681-5$$a10.1038/s41418-023-01153-w$$gVol. 30, no. 5, p. 1097 - 1154$$n5$$p1097 - 1154$$tCell death and differentiation$$v30$$x1350-9047$$y2023
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