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@ARTICLE{Kloske:258684,
author = {Kloske, Courtney M and Barnum, Christopher J and Batista,
Andre F and Bradshaw, Elizabeth M and Brickman, Adam M and
Bu, Guojun and Dennison, Jessica and Gearon, Mary D and
Goate, Alison M and Haass, Christian and Heneka, Michael and
Hu, William T and Huggins, Lenique K L and Jones, Nahdia S
and Koldamova, Radosveta and Lemere, Cynthia A and Liddelow,
Shane A and Marcora, Edoardo and Marsh, Samuel E and
Nielsen, Henrietta M and Petersen, Kellen K and Petersen,
Melissa and Piña-Escudero, Stefanie D and Qiu, Wei Qiao and
Quiroz, Yakeel T and Reiman, Eric and Sexton, Claire and
Tansey, Malú Gámez and Tcw, Julia and Teunissen, Charlotte
E and Tijms, Betty M and van der Kant, Rik and Wallings,
Rebecca and Weninger, Stacie C and Wharton, Whitney and
Wilcock, Donna M and Wishard, Tyler James and Worley, Susan
L and Zetterberg, Henrik and Carrillo, Maria C},
title = {{APOE} and immunity: {R}esearch highlights.},
journal = {Alzheimer's and dementia},
volume = {19},
number = {6},
issn = {1552-5260},
address = {Hoboken, NJ},
publisher = {Wiley},
reportid = {DZNE-2023-00657},
pages = {2677 - 2696},
year = {2023},
abstract = {At the Alzheimer's Association's APOE and Immunity virtual
conference, held in October 2021, leading neuroscience
experts shared recent research advances on and inspiring
insights into the various roles that both the apolipoprotein
E gene (APOE) and facets of immunity play in
neurodegenerative diseases, including Alzheimer's disease
and other dementias.The meeting brought together more than
1200 registered attendees from 62 different countries,
representing the realms of academia and industry.During the
4-day meeting, presenters illuminated aspects of the
cross-talk between APOE and immunity, with a focus on the
roles of microglia, triggering receptor expressed on myeloid
cells 2 (TREM2), and components of inflammation (e.g., tumor
necrosis factor α [TNFα]).This manuscript emphasizes the
importance of diversity in current and future research and
presents an integrated view of innate immune functions in
Alzheimer's disease as well as related promising directions
in drug development.},
subtyp = {Review Article},
keywords = {Humans / Alzheimer Disease: pathology / Microglia:
pathology / Inflammation / Apolipoproteins E: genetics /
APOE (Other) / Alzheimer's disease (Other) / TREM2 (Other) /
amyloid beta (Other) / astrocytes (Other) / biomarkers
(Other) / dementia (Other) / inflammation (Other) /
microglia (Other) / neurodegeneration (Other) / neuroglia
(Other) / neuroinflammation (Other) / tau (Other) /
Apolipoproteins E (NLM Chemicals)},
cin = {AG Haass},
ddc = {610},
cid = {I:(DE-2719)1110007},
pnm = {352 - Disease Mechanisms (POF4-352)},
pid = {G:(DE-HGF)POF4-352},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:36975090},
doi = {10.1002/alz.13020},
url = {https://pub.dzne.de/record/258684},
}