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@ARTICLE{Kloske:258684,
      author       = {Kloske, Courtney M and Barnum, Christopher J and Batista,
                      Andre F and Bradshaw, Elizabeth M and Brickman, Adam M and
                      Bu, Guojun and Dennison, Jessica and Gearon, Mary D and
                      Goate, Alison M and Haass, Christian and Heneka, Michael and
                      Hu, William T and Huggins, Lenique K L and Jones, Nahdia S
                      and Koldamova, Radosveta and Lemere, Cynthia A and Liddelow,
                      Shane A and Marcora, Edoardo and Marsh, Samuel E and
                      Nielsen, Henrietta M and Petersen, Kellen K and Petersen,
                      Melissa and Piña-Escudero, Stefanie D and Qiu, Wei Qiao and
                      Quiroz, Yakeel T and Reiman, Eric and Sexton, Claire and
                      Tansey, Malú Gámez and Tcw, Julia and Teunissen, Charlotte
                      E and Tijms, Betty M and van der Kant, Rik and Wallings,
                      Rebecca and Weninger, Stacie C and Wharton, Whitney and
                      Wilcock, Donna M and Wishard, Tyler James and Worley, Susan
                      L and Zetterberg, Henrik and Carrillo, Maria C},
      title        = {{APOE} and immunity: {R}esearch highlights.},
      journal      = {Alzheimer's and dementia},
      volume       = {19},
      number       = {6},
      issn         = {1552-5260},
      address      = {Hoboken, NJ},
      publisher    = {Wiley},
      reportid     = {DZNE-2023-00657},
      pages        = {2677 - 2696},
      year         = {2023},
      abstract     = {At the Alzheimer's Association's APOE and Immunity virtual
                      conference, held in October 2021, leading neuroscience
                      experts shared recent research advances on and inspiring
                      insights into the various roles that both the apolipoprotein
                      E gene (APOE) and facets of immunity play in
                      neurodegenerative diseases, including Alzheimer's disease
                      and other dementias.The meeting brought together more than
                      1200 registered attendees from 62 different countries,
                      representing the realms of academia and industry.During the
                      4-day meeting, presenters illuminated aspects of the
                      cross-talk between APOE and immunity, with a focus on the
                      roles of microglia, triggering receptor expressed on myeloid
                      cells 2 (TREM2), and components of inflammation (e.g., tumor
                      necrosis factor α [TNFα]).This manuscript emphasizes the
                      importance of diversity in current and future research and
                      presents an integrated view of innate immune functions in
                      Alzheimer's disease as well as related promising directions
                      in drug development.},
      subtyp        = {Review Article},
      keywords     = {Humans / Alzheimer Disease: pathology / Microglia:
                      pathology / Inflammation / Apolipoproteins E: genetics /
                      APOE (Other) / Alzheimer's disease (Other) / TREM2 (Other) /
                      amyloid beta (Other) / astrocytes (Other) / biomarkers
                      (Other) / dementia (Other) / inflammation (Other) /
                      microglia (Other) / neurodegeneration (Other) / neuroglia
                      (Other) / neuroinflammation (Other) / tau (Other) /
                      Apolipoproteins E (NLM Chemicals)},
      cin          = {AG Haass},
      ddc          = {610},
      cid          = {I:(DE-2719)1110007},
      pnm          = {352 - Disease Mechanisms (POF4-352)},
      pid          = {G:(DE-HGF)POF4-352},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:36975090},
      doi          = {10.1002/alz.13020},
      url          = {https://pub.dzne.de/record/258684},
}