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@ARTICLE{Jiang:258690,
author = {Jiang, Xueyan and Hu, Xiaochen and Daamen, Marcel and Wang,
Xiaoqi and Fan, Chunqiu and Meiberth, Dix and Spottke,
Annika and Roeske, Sandra and Fliessbach, Klaus and Spruth,
Eike Jakob and Altenstein, Slawek and Lohse, Andrea and
Hansen, Niels and Glanz, Wenzel and Incesoy, Enise and
Dobisch, Laura and Janowitz, Daniel and Rauchmann, Boris
Stephan and Ramirez, Alfredo and Kilimann, Ingo and Munk,
Matthias and Wang, Xiao and Schneider, Luisa-Sophie and
Gabelin, Tatjana and Roy, Nina and Wolfsgruber, Steffen and
Kleineidam, Luca and Hetzer, Stefan and Dechent, Peter and
Ewers, Michael and Scheffler, Klaus and Amthauer, Holger and
Buchert, Ralph and Essler, Markus and Drzezga, Alexander and
Rominger, Axel and Krause, Bernd J and Reimold, Matthias and
Priller, Josef and Schneider, Anja and Wiltfang, Jens and
Buerger, Katharina and Perneczky, Robert and Teipel, Stefan
and Laske, Christoph and Peters, Oliver and Düzel, Emrah
and Wagner, Michael and Jiang, Jiehui and Jessen, Frank and
Boecker, Henning and Han, Ying},
title = {{A}ltered limbic functional connectivity in individuals
with subjective cognitive decline: {C}onverging and
diverging findings across {C}hinese and {G}erman cohorts.},
journal = {Alzheimer's and dementia},
volume = {19},
number = {11},
issn = {1552-5260},
address = {Hoboken, NJ},
publisher = {Wiley},
reportid = {DZNE-2023-00663},
pages = {4922 - 4934},
year = {2023},
abstract = {It remains unclear whether functional brain networks are
consistently altered in individuals with subjective
cognitive decline (SCD) of diverse ethnic and cultural
backgrounds and whether the network alterations are
associated with an amyloid burden.Cross-sectional
resting-state functional magnetic resonance imaging
connectivity (FC) and amyloid-positron emission tomography
(PET) data from the Chinese Sino Longitudinal Study on
Cognitive Decline and German DZNE Longitudinal Cognitive
Impairment and Dementia cohorts were analyzed.Limbic FC,
particularly hippocampal connectivity with right insula, was
consistently higher in SCD than in controls, and correlated
with SCD-plus features. Smaller SCD subcohorts with PET
showed inconsistent amyloid positivity rates and FC-amyloid
associations across cohorts.Our results suggest an early
adaptation of the limbic network in SCD, which may reflect
increased awareness of cognitive decline, irrespective of
amyloid pathology. Different amyloid positivity rates may
indicate a heterogeneous underlying etiology in Eastern and
Western SCD cohorts when applying current research criteria.
Future studies should identify culture-specific features to
enrich preclinical Alzheimer's disease in non-Western
populations.Common limbic hyperconnectivity across Chinese
and German subjective cognitive decline (SCD) cohorts was
observed. Limbic hyperconnectivity may reflect awareness of
cognition, irrespective of amyloid load. Further
cross-cultural harmonization of SCD regarding Alzheimer's
disease pathology is required.},
keywords = {European People / Humans / Alzheimer Disease: pathology /
Amyloid beta-Peptides: metabolism / Brain: pathology /
Cognitive Dysfunction / Cross-Sectional Studies / East Asian
People / Magnetic Resonance Imaging / Positron-Emission
Tomography / Amyloid beta-Peptides (NLM Chemicals) /
Centiloid (Other) / amyloid deposition (Other) /
cross-cultural harmonization (Other) / functional
connectivity (Other) / hippocampus (Other) / insula (Other)
/ subjective cognitive decline (Other)},
cin = {Patient Studies Bonn / AG Jessen / AG Boecker / Clinical
Research Platform (CRP) / AG Wagner / AG Endres / AG Düzel
/ AG Priller / AG Schneider / AG Wiltfang / AG Levin / AG
Dichgans / AG Teipel / AG Gasser / AG Peters},
ddc = {610},
cid = {I:(DE-2719)1011101 / I:(DE-2719)1011102 /
I:(DE-2719)1011202 / I:(DE-2719)1011401 / I:(DE-2719)1011201
/ I:(DE-2719)1811005 / I:(DE-2719)5000006 /
I:(DE-2719)5000007 / I:(DE-2719)1011305 / I:(DE-2719)1410006
/ I:(DE-2719)1111016 / I:(DE-2719)5000022 /
I:(DE-2719)1510100 / I:(DE-2719)1210000 /
I:(DE-2719)5000000},
pnm = {353 - Clinical and Health Care Research (POF4-353) / 352 -
Disease Mechanisms (POF4-352)},
pid = {G:(DE-HGF)POF4-353 / G:(DE-HGF)POF4-352},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:37070734},
doi = {10.1002/alz.13068},
url = {https://pub.dzne.de/record/258690},
}