TY - JOUR AU - Ramachandran, Swetha AU - Grozdanov, Veselin AU - Leins, Bianca AU - Kandler, Katharina AU - Witzel, Simon AU - Mulaw, Medhanie AU - Ludolph, Albert C. AU - Weishaupt, Jochen H. AU - Danzer, Karin M. TI - Low T-cell reactivity to TDP-43 peptides in ALS JO - Frontiers in immunology VL - 14 SN - 1664-3224 CY - Lausanne PB - Frontiers Media M1 - DZNE-2023-00775 SP - 1193507 PY - 2023 AB - Dysregulation of the immune system in amyotrophic lateral sclerosis (ALS) includes changes in T-cells composition and infiltration of T cells in the brain and spinal cord. Recent studies have shown that cytotoxic T cells can directly induce motor neuron death in a mouse model of ALS and that T cells from ALS patients are cytotoxic to iPSC-derived motor neurons from ALS patients. Furthermore, a clonal expansion to unknown epitope(s) was recently found in familial ALS and increased peripheral and intrathecal activation of cytotoxic CD8+ T cells in sporadic ALS.Here, we show an increased activation of peripheral T cells from patients with sporadic ALS by IL-2 treatment, suggesting an increase of antigen-experienced T cells in ALS blood. However, a putative antigen for T-cell activation in ALS has not yet been identified. Therefore, we investigated if peptides derived from TDP-43, a key protein in ALS pathogenesis, can act as epitopes for antigen-mediated activation of human T cells by ELISPOT and flow cytometry. We found that TDP-43 peptides induced only a weak MHCI or MHCII-restricted activation of both naïve and antigen-experienced T cells from healthy controls and ALS patients. Interestingly, we found less activation in T cells from ALS patients to TDP-43 and control stimuli. Furthermore, we found no change in the levels of naturally occurring auto-antibodies against full-length TDP-43 in ALS.Our data suggests a general increase in antigen-experienced T cells in ALS blood, measured by in-vitro culture with IL-2 for 14 days. Furthermore, it suggests that TDP-43 is a weak autoantigen. KW - Humans KW - Amyotrophic Lateral Sclerosis: metabolism KW - CD8-Positive T-Lymphocytes: metabolism KW - DNA-Binding Proteins: metabolism KW - Interleukin-2 KW - DNA-Binding Proteins (NLM Chemicals) KW - T cells (Other) KW - TDP-43 (Other) KW - amyotrophic lateral sclerosis (Other) KW - autoantibody (Other) KW - autoantigen (Other) KW - Interleukin-2 (NLM Chemicals) KW - TARDBP protein, human (NLM Chemicals) LB - PUB:(DE-HGF)16 C2 - pmc:PMC10401033 C6 - pmid:37545536 DO - DOI:10.3389/fimmu.2023.1193507 UR - https://pub.dzne.de/record/259703 ER -