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@ARTICLE{Salomoni:266479,
author = {Salomoni, Paolo and Flanagan, Adrienne M. and Cottone,
Lucia},
title = {({B}){O}n(e)-cohistones and the epigenetic alterations at
the root of bone cancer},
journal = {Cell death and differentiation},
volume = {32},
number = {1},
issn = {1350-9047},
address = {London},
publisher = {Macmillan},
reportid = {DZNE-2023-01164},
pages = {66 - 77},
year = {2025},
abstract = {Identification of mutations in histones in a number of
human neoplasms and developmental syndromes represents the
most compelling evidence to date for a causal role of
epigenetic perturbations in human disease. In most cases,
these mutations have gain of function properties that cause
deviation from normal developmental processes leading to
embryo defects and/or neoplastic transformation. These
exciting discoveries represent a step-change in our
understanding of the role of chromatin (dys)regulation in
development and disease. However, the mechanisms of action
of oncogenic histone mutations (oncohistones) remain only
partially understood. Here, we critically assess existing
literature on oncohistones focussing mainly on bone
neoplasms. We show how it is possible to draw parallels with
some of the cell-autonomous mechanisms of action described
in paediatric brain cancer, although the functions of
oncohistones in bone tumours remain under-investigated. In
this respect, it is becoming clear that histone mutations
targeting the same residues display, at least in part,
tissue-specific oncogenic mechanisms. Furthermore, it is
emerging that cancer cells carrying oncohistones can modify
the surrounding microenvironment to support growth and/or
alter differentiation trajectories. A better understanding
of oncohistone function in different neoplasms provide
potential for identification of signalling that could be
targeted therapeutically. Finally, we discuss some of the
main concepts and future directions in this research area,
while also drawing possible connections and parallels with
other cancer epigenetic mechanisms.},
subtyp = {Review Article},
keywords = {Humans / Epigenesis, Genetic / Bone Neoplasms: genetics /
Bone Neoplasms: metabolism / Bone Neoplasms: pathology /
Histones: metabolism / Mutation / Animals},
cin = {AG Salomoni},
ddc = {610},
cid = {I:(DE-2719)1013032},
pnm = {352 - Disease Mechanisms (POF4-352)},
pid = {G:(DE-HGF)POF4-352},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:37828086},
pmc = {pmc:PMC11748643},
doi = {10.1038/s41418-023-01227-9},
url = {https://pub.dzne.de/record/266479},
}