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@ARTICLE{Vallucci:266788,
author = {Vallucci, Maeva and Boutin, Jean A and Janda, Elzbieta and
Blandel, Florence and Musgrove, Ruth and Di Monte, Donato
and Ferry, Gilles and Michel, Patrick P and Hirsch, Etienne
C},
title = {{T}he specific {NQO}2 inhibitor, {S}29434, only marginally
improves the survival of dopamine neurons in
{MPTP}-intoxicated mice.},
journal = {Journal of neural transmission},
volume = {131},
number = {1},
issn = {0375-9245},
address = {Wien [u.a.]},
publisher = {Springer},
reportid = {DZNE-2024-00046},
pages = {1 - 11},
year = {2024},
abstract = {Over the years, evidence has accumulated on a possible
contributive role of the cytosolic quinone reductase NQO2 in
models of dopamine neuron degeneration induced by
parkinsonian toxin, but most of the data have been obtained
in vitro. For this reason, we asked the question whether
NQO2 is involved in the in vivo toxicity of MPTP, a
neurotoxin classically used to model Parkinson
disease-induced neurodegeneration. First, we show that NQO2
is expressed in mouse substantia nigra dopaminergic cell
bodies and in human dopaminergic SH-SY5Y cells as well. A
highly specific NQO2 inhibitor, S29434, was able to reduce
MPTP-induced cell death in a co-culture system of SH-SY5Y
cells with astrocytoma U373 cells but was inactive in
SH-SY5Y monocultures. We found that S29434 only marginally
prevents substantia nigra tyrosine hydroxylase+ cell loss
after MPTP intoxication in vivo. The compound produced a
slight increase of dopaminergic cell survival at day 7 and
21 following MPTP treatment, especially with 1.5 and 3 mg/kg
dosage regimen. The rescue effect did not reach statistical
significance (except for one experiment at day 7) and tended
to decrease with the 4.5 mg/kg dose, at the latest time
point. Despite the lack of robust protective activity of the
inhibitor of NQO2 in the mouse MPTP model, we cannot rule
out a possible role of the enzyme in parkinsonian
degeneration, particularly because it is substantially
expressed in dopaminergic neurons.},
keywords = {Mice / Humans / Animals / Dopaminergic Neurons: metabolism
/ MPTP Poisoning / Neuroblastoma / Substantia Nigra:
metabolism / Dopamine: metabolism / Mice, Inbred C57BL /
Disease Models, Animal / MPTP (Other) / NQO2 (Other) /
Neuroprotection (Other) / Parkinson disease (Other) / S29434
(Other) / NRH - quinone oxidoreductase2 (NLM Chemicals) /
N-(2-(2-methoxy-6H-dipyrido(2,3-a-3,2-e)pyrrolizin-11-yl)ethyl)-2-furamide
(NLM Chemicals) / Dopamine (NLM Chemicals)},
cin = {AG Di Monte},
ddc = {610},
cid = {I:(DE-2719)1013008},
pnm = {352 - Disease Mechanisms (POF4-352)},
pid = {G:(DE-HGF)POF4-352},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:37851107},
doi = {10.1007/s00702-023-02709-3},
url = {https://pub.dzne.de/record/266788},
}