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@ARTICLE{Teipel:267052,
author = {Teipel, Stefan and Dyrba, Martin and Kleineidam, Luca and
Brosseron, Frederic and Levin, Fedor and Bruno, Davide and
Buerger, Katharina and Cosma, Nicoleta and Schneider,
Luisa-Sophie and Düzel, Emrah and Glanz, Wenzel and
Fliessbach, Klaus and Janowitz, Daniel and Kilimann, Ingo
and Laske, Christoph and Munk, Matthias H and Maier,
Franziska and Peters, Oliver and Pomara, Nunzio and
Perneczky, Robert and Rauchmann, Boris Stephan and Priller,
Josef and Ramirez, Alfredo and Roy, Nina and Schneider, Anja
and Spottke, Annika and Spruth, Eike Jakob and Roeske,
Sandra and Wagner, Michael and Wiltfang, Jens and
Wolfsgruber, Steffen and Bartels, Claudia and Jessen, Frank
and Heneka, Michael T and group, DELCODE study},
collaboration = {Initiative, the Alzheimer´s Disease Neuroimaging},
title = {{A}ssociation of latent factors of neuroinflammation with
{A}lzheimer's disease pathology and longitudinal cognitive
decline.},
journal = {Alzheimer's $\&$ dementia / Diagnosis, assessment $\&$
disease monitoring},
volume = {16},
number = {1},
issn = {2352-8729},
address = {Hoboken, NJ},
publisher = {Wiley},
reportid = {DZNE-2024-00061},
pages = {e12510},
year = {2024},
abstract = {We investigated the association of inflammatory mechanisms
with markers of Alzheimer's disease (AD) pathology and rates
of cognitive decline in the AD spectrum.We studied 296 cases
from the Deutsches Zentrum für Neurodegenerative
Erkrankungen Longitudinal Cognitive Impairment and Dementia
Study (DELCODE) cohort, and an extension cohort of 276 cases
of the Alzheimer's Disease Neuroimaging Initiative study.
Using Bayesian confirmatory factor analysis, we constructed
latent factors for synaptic integrity, microglia,
cerebrovascular endothelial function, cytokine/chemokine,
and complement components of the inflammatory response using
a set of inflammatory markers in cerebrospinal fluid.We
found strong evidence for an association of synaptic
integrity, microglia response, and cerebrovascular
endothelial function with a latent factor of AD pathology
and with rates of cognitive decline. We found evidence
against an association of complement and cytokine/chemokine
factors with AD pathology and rates of cognitive
decline.Latent factors provided access to directly
unobservable components of the neuroinflammatory response
and their association with AD pathology and cognitive
decline.},
keywords = {amyloid (Other) / chemokine factors (Other) / complement
(Other) / endothelial function (Other) / microglia (Other) /
structural equation models (Other) / tau (Other)},
cin = {AG Heneka ; AG Heneka / AG Teipel / AG Wagner / Clinical
Research (Munich) / AG Düzel / Patient Studies Bonn / AG
Gasser / AG Peters / AG Priller / AG Simons / AG Dichgans /
AG Spottke / AG Schneider / AG Wiltfang / AG Jessen},
ddc = {610},
cid = {I:(DE-2719)1011303 / I:(DE-2719)1510100 /
I:(DE-2719)1011201 / I:(DE-2719)1111015 / I:(DE-2719)5000006
/ I:(DE-2719)1011101 / I:(DE-2719)1210000 /
I:(DE-2719)5000000 / I:(DE-2719)5000007 / I:(DE-2719)1110008
/ I:(DE-2719)5000022 / I:(DE-2719)1011103 /
I:(DE-2719)1011305 / I:(DE-2719)1410006 /
I:(DE-2719)1011102},
pnm = {353 - Clinical and Health Care Research (POF4-353)},
pid = {G:(DE-HGF)POF4-353},
experiment = {EXP:(DE-2719)DELCODE-20140101},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:38213951},
pmc = {pmc:PMC10781650},
doi = {10.1002/dad2.12510},
url = {https://pub.dzne.de/record/267052},
}