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001     268320
005     20250127091801.0
024 7 _ |a pmc:PMC11492925
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024 7 _ |a 10.1038/s41591-023-02768-9
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024 7 _ |a pmid:38287169
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024 7 _ |a 1078-8956
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024 7 _ |a 1546-170X
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037 _ _ |a DZNE-2024-00202
041 _ _ |a English
082 _ _ |a 610
100 1 _ |a Jucker, Mathias
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245 _ _ |a Evidence for iatrogenic transmission of Alzheimer's disease.
260 _ _ |a New York, NY
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520 _ _ |a Credible evidence suggests that, under extraordinary circumstances, Alzheimer’s disease may be transmitted by a prion-like mechanism — yielding insights into both the basic biology of this neurodegenerative disorder and strategies for early prevention.
536 _ _ |a 352 - Disease Mechanisms (POF4-352)
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650 _ 7 |a Amyloid beta-Peptides
|2 NLM Chemicals
650 _ 2 |a Animals
|2 MeSH
650 _ 2 |a Humans
|2 MeSH
650 _ 2 |a Alzheimer Disease: etiology
|2 MeSH
650 _ 2 |a Amyloid beta-Peptides
|2 MeSH
650 _ 2 |a Disease Models, Animal
|2 MeSH
650 _ 2 |a Iatrogenic Disease
|2 MeSH
700 1 _ |a Walker, Lary C
|b 1
773 _ _ |a 10.1038/s41591-023-02768-9
|g Vol. 30, no. 2, p. 344 - 345
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|t Nature medicine
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856 4 _ |u https://pub.dzne.de/record/268320/files/DZNE-2024-00202_Restricted.pdf
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910 1 _ |a Deutsches Zentrum für Neurodegenerative Erkrankungen
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