%0 Journal Article
%A Shrouder, Joshua James
%A Calandra, Gian-Marco
%A Filser, Severin
%A Varga, Daniel Peter
%A Besson-Girard, Simon
%A Mamrak, Uta
%A Dorok, Maximilian
%A Bulut-Impraim, Buket
%A Seker, Fatma Burcu
%A Gesierich, Benno
%A Laredo, Fabio
%A Wehn, Antonia Clarissa
%A Khalin, Igor
%A Bayer, Patrick
%A Liesz, Arthur
%A Gökce, Ozgun
%A Plesnila, Nikolaus
%T Continued dysfunction of capillary pericytes promotes no-reflow after experimental stroke in vivo.
%J Brain
%V 147
%N 3
%@ 0006-8950
%C Oxford
%I Oxford Univ. Press
%M DZNE-2024-00246
%P 1057 - 1074
%D 2024
%X Incomplete reperfusion of the microvasculature ('no-reflow') after ischaemic stroke damages salvageable brain tissue. Previous ex vivo studies suggest pericytes are vulnerable to ischaemia and may exacerbate no-reflow, but the viability of pericytes and their association with no-reflow remains under-explored in vivo. Using longitudinal in vivo two-photon single-cell imaging over 7 days, we showed that 87
%K Humans
%K Stroke
%K Pericytes: physiology
%K Brain Ischemia
%K Ischemic Stroke
%K Cerebral Infarction
%K cerebral ischaemia (Other)
%K ischaemic stroke (Other)
%K no-reflow (Other)
%K pericytes (Other)
%K reperfusion (Other)
%F PUB:(DE-HGF)16
%9 Journal Article
%$ pmid:38153327
%R 10.1093/brain/awad401
%U https://pub.dzne.de/record/268500