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@ARTICLE{Radke:268524,
author = {Radke, Josefine and Meinhardt, Jenny and Aschman, Tom and
Chua, Robert Lorenz and Farztdinov, Vadim and Lukassen,
Sören and Ten, Foo Wei and Friebel, Ekaterina and Ishaque,
Naveed and Franz, Jonas and Huhle, Valerie Helena and
Mothes, Ronja and Peters, Kristin and Thomas, Carolina and
Schneeberger, Shirin and Schumann, Elisa and Kawelke, Leona
and Jünger, Julia and Horst, Viktor and Streit, Simon and
von Manitius, Regina and Körtvelyessy, Peter and Vielhaber,
Stefan and Reinhold, Dirk and Hauser, Anja E and Osterloh,
Anja and Enghard, Philipp and Ihlow, Jana and Elezkurtaj,
Sefer and Horst, David and Kurth, Florian and Müller,
Marcel A and Gassen, Nils C and Melchert, Julia and Jechow,
Katharina and Timmermann, Bernd and Fernandez-Zapata, Camila
and Böttcher, Chotima and Stenzel, Werner and Krüger, Elke
and Landthaler, Markus and Wyler, Emanuel and Corman, Victor
and Stadelmann, Christine and Ralser, Markus and Eils,
Roland and Heppner, Frank L and Mülleder, Michael and
Conrad, Christian and Radbruch, Helena},
title = {{P}roteomic and transcriptomic profiling of brainstem,
cerebellum and olfactory tissues in early- and late-phase
{COVID}-19.},
journal = {Nature neuroscience},
volume = {27},
number = {3},
issn = {1097-6256},
address = {New York, NY},
publisher = {Nature America},
reportid = {DZNE-2024-00265},
pages = {409 - 420},
year = {2024},
abstract = {Neurological symptoms, including cognitive impairment and
fatigue, can occur in both the acute infection phase of
coronavirus disease 2019 (COVID-19) and at later stages, yet
the mechanisms that contribute to this remain unclear. Here
we profiled single-nucleus transcriptomes and proteomes of
brainstem tissue from deceased individuals at various stages
of COVID-19. We detected an inflammatory type I interferon
response in acute COVID-19 cases, which resolves in the late
disease phase. Integrating single-nucleus RNA sequencing and
spatial transcriptomics, we could localize two patterns of
reaction to severe systemic inflammation, one neuronal with
a direct focus on cranial nerve nuclei and a separate
diffuse pattern affecting the whole brainstem. The latter
reflects a bystander effect of the respiratory infection
that spreads throughout the vascular unit and alters the
transcriptional state of mainly oligodendrocytes, microglia
and astrocytes, while alterations of the brainstem nuclei
could reflect the connection of the immune system and the
central nervous system via, for example, the vagus nerve.
Our results indicate that even without persistence of severe
acute respiratory syndrome coronavirus 2 in the central
nervous system, local immune reactions are prevailing,
potentially causing functional disturbances that contribute
to neurological complications of COVID-19.},
keywords = {Humans / COVID-19: genetics / Proteomics / Brain Stem /
Cerebellum / Gene Expression Profiling},
cin = {AG Heppner},
ddc = {610},
cid = {I:(DE-2719)1810007},
pnm = {353 - Clinical and Health Care Research (POF4-353)},
pid = {G:(DE-HGF)POF4-353},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:38366144},
doi = {10.1038/s41593-024-01573-y},
url = {https://pub.dzne.de/record/268524},
}