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@ARTICLE{Papazoglou:269686,
author = {Papazoglou, Anna and Henseler, Christina and Weickhardt,
Sandra and Daubner, Johanna and Schiffer, Teresa and Broich,
Karl and Hescheler, Jürgen and Sachinidis, Agapios and
Ehninger, Dan and Haenisch, Britta and Weiergräber, Marco},
title = {{S}ex-specific cortical, hippocampal and thalamic whole
genome transcriptome data from controls and a {G}72
schizophrenia mouse model.},
journal = {BMC Research Notes},
volume = {17},
number = {1},
issn = {1756-0500},
address = {London},
publisher = {[Verlag nicht ermittelbar]},
reportid = {DZNE-2024-00600},
pages = {143},
year = {2024},
abstract = {The G72 mouse model of schizophrenia represents a
well-known model that was generated to meet the main
translational criteria of isomorphism, homology and
predictability of schizophrenia to a maximum extent. In
order to get a more detailed view of the complex
etiopathogenesis of schizophrenia, whole genome
transcriptome studies turn out to be indispensable. Here we
carried out microarray data collection based on RNA
extracted from the retrosplenial cortex, hippocampus and
thalamus of G72 transgenic and wild-type control mice.
Experimental animals were age-matched and importantly, both
sexes were considered separately.The isolated RNA from all
three brain regions was purified, quantified und quality
controlled before initiation of the hybridization procedure
with SurePrint G3 Mouse Gene Expression v2 8 × 60 K
microarrays. Following immunofluorescent measurement und
preprocessing of image data, raw transcriptome data from G72
mice and control animals were extracted and uploaded in a
public database. Our data allow insight into significant
alterations in gene transcript levels in G72 mice and enable
the reader/user to perform further complex analyses to
identify potential age-, sex- and brain-region-specific
alterations in transcription profiles and related pathways.
The latter could facilitate biomarker identification and
drug research and development in schizophrenia research.},
keywords = {Animals / Schizophrenia: genetics / Schizophrenia:
metabolism / Hippocampus: metabolism / Male / Female / Mice
/ Disease Models, Animal / Transcriptome: genetics /
Cerebral Cortex: metabolism / Cerebral Cortex: pathology /
Thalamus: metabolism / Mice, Transgenic / Gene Expression
Profiling: methods / Sex Factors / Brain (Other) / Fold
change (Other) / Hippocampus (Other) / Hybridization (Other)
/ Microarray (Other) / RNA (Other) / Retrosplenial cortex
(Other) / Schizophrenia (Other) / Thalamus (Other) /
Transcriptome (Other)},
cin = {AG Ehninger / AG Hänisch},
ddc = {570},
cid = {I:(DE-2719)1013005 / I:(DE-2719)1013010},
pnm = {352 - Disease Mechanisms (POF4-352) / 354 - Disease
Prevention and Healthy Aging (POF4-354)},
pid = {G:(DE-HGF)POF4-352 / G:(DE-HGF)POF4-354},
typ = {PUB:(DE-HGF)16},
pmc = {pmc:PMC11110308},
pubmed = {pmid:38773625},
doi = {10.1186/s13104-024-06799-4},
url = {https://pub.dzne.de/record/269686},
}