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@ARTICLE{Aziz:270185,
author = {Aziz, N. Ahmad and Santos, Marina and Breteler, Monique},
title = {{T}he association between {SARS}-{C}o{V}-2 infection and
neuronal damage: {A} population-based nested case-control
study},
reportid = {DZNE-2024-00756},
year = {2021},
abstract = {Objective: To assess whether severe acute respiratory
syndrome coronavirus-2 (SARS-CoV-2) infection is associated
with changes in plasma levels of neurofilament light chain
(NfL), an extremely sensitive marker of neuroaxonal damage,
in community-dwelling individuals.Setting: This study was
embedded within the Rhineland Study, an ongoing
community-based cohort study in Bonn, GermanyDesign:
Cross-sectional nested case-control study.Participants:
Participants were selected based on results from a
previously conducted seroprevalence survey within the
framework of the Rhineland Study. Cases were defined as
those individuals who had had two positive confirmatory test
results, including a recombinant spike-based
immunofluorescence assay and a plaque reduction
neutralization test (N=21). As controls, a random sample of
individuals with a negative ELISA test result (Controls I,
N=1117), and those with a borderline or positive ELISA test
result who failed confirmatory testing (Controls II, N=68),
were selected.Outcome measures: Plasma levels of NfL at the
time of measurement, as well as change in plasma NfL levels
compared to previously measured pre-pandemic levelsResults:
After adjustment for age, sex and batch effects,
serologically confirmed SARS-CoV-2 infection was neither
associated with cross-sectional NfL levels, nor with the
magnitude of change from pre-pandemic levels, compared to
either of the two control groups. Similarly, after
adjustment for age, sex and batch effects, self-reported
neurological symptoms – including altered sense of smell
or taste, headache, myalgia and fever – were not
associated with changes in NfL levels in participants with a
serologically confirmed SARS-CoV-2 infection (all p ≥
0.56).Conclusions: Our findings indicate that
mild-to-moderate coronavirus disease-19 is unlikely to be
associated with a clinically relevant degree of neuroaxonal
damage, even in those cases associated with neurological
symptoms.},
cin = {AG Aziz / AG Breteler},
cid = {I:(DE-2719)5000071 / I:(DE-2719)1012001},
pnm = {354 - Disease Prevention and Healthy Aging (POF4-354)},
pid = {G:(DE-HGF)POF4-354},
experiment = {EXP:(DE-2719)Rhineland Study-20190321},
typ = {PUB:(DE-HGF)25},
doi = {10.1101/2021.09.02.21263019},
url = {https://pub.dzne.de/record/270185},
}