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@ARTICLE{Arner:271882,
author = {Arner, Anja and Ettinger, Andreas and Blaser, Bradley Wayne
and Schmid, Bettina and Jeremias, Irmela and Rostam, Nadia
and Binder-Blaser, Vera},
title = {{I}n vivo monitoring of leukemia-niche interactions in a
zebrafish xenograft model.},
journal = {PLOS ONE},
volume = {19},
number = {8},
issn = {1932-6203},
address = {San Francisco, California, US},
publisher = {PLOS},
reportid = {DZNE-2024-01094},
pages = {e0309415},
year = {2024},
abstract = {Acute lymphoblastic leukemia (ALL) is the most common type
of malignancy in children. ALL prognosis after initial
diagnosis is generally good; however, patients suffering
from relapse have a poor outcome. The tumor microenvironment
is recognized as an important contributor to relapse, yet
the cell-cell interactions involved are complex and
difficult to study in traditional experimental models. In
the present study, we established an innovative larval
zebrafish xenotransplantation model, that allows the
analysis of leukemic cells (LCs) within an orthotopic niche
using time-lapse microscopic and flow cytometric approaches.
LCs homed, engrafted and proliferated within the
hematopoietic niche at the time of transplant, the caudal
hematopoietic tissue (CHT). A specific dissemination pattern
of LCs within the CHT was recorded, as they extravasated
over time and formed clusters close to the dorsal aorta.
Interactions of LCs with macrophages and endothelial cells
could be quantitatively characterized. This zebrafish model
will allow the quantitative analysis of LCs in a functional
and complex microenvironment, to study mechanisms of niche
mediated leukemogenesis, leukemia maintenance and relapse
development.},
keywords = {Animals / Zebrafish / Tumor Microenvironment / Precursor
Cell Lymphoblastic Leukemia-Lymphoma: pathology / Humans /
Disease Models, Animal / Cell Communication / Heterografts /
Stem Cell Niche / Cell Line, Tumor / Endothelial Cells:
pathology / Macrophages: pathology / Macrophages: metabolism
/ Transplantation, Heterologous},
cin = {AG Schmid München},
ddc = {610},
cid = {I:(DE-2719)1140002},
pnm = {352 - Disease Mechanisms (POF4-352)},
pid = {G:(DE-HGF)POF4-352},
typ = {PUB:(DE-HGF)16},
pmc = {pmc:PMC11364250},
pubmed = {pmid:39213296},
doi = {10.1371/journal.pone.0309415},
url = {https://pub.dzne.de/record/271882},
}