% IMPORTANT: The following is UTF-8 encoded. This means that in the presence
% of non-ASCII characters, it will not work with BibTeX 0.99 or older.
% Instead, you should use an up-to-date BibTeX implementation like “bibtex8” or
% “biber”.
@ARTICLE{Skrobot:271972,
author = {Skrobot, Matej and Sa, Rafael De and Walter, Josefine and
Vogt, Arend and Paulat, Raik and Lips, Janet and Mosch,
Larissa and Mueller, Susanne and Dominiak, Sina and Sachdev,
Robert and Boehm-Sturm, Philipp and Dirnagl, Ulrich and
Endres, Matthias and Harms, Christoph and Wenger, Nikolaus},
title = {{R}efined movement analysis in the staircase test reveals
differential motor deficits in mouse models of stroke.},
journal = {Journal of cerebral blood flow $\&$ metabolism},
volume = {44},
number = {9},
issn = {0271-678X},
address = {London},
publisher = {Sage},
reportid = {DZNE-2024-01114},
pages = {1551 - 1564},
year = {2024},
abstract = {Accurate assessment of post-stroke deficits is crucial in
translational research. Recent advances in machine learning
offer precise quantification of rodent motor behavior
post-stroke, yet detecting lesion-specific upper extremity
deficits remains unclear. Employing proximal middle cerebral
artery occlusion (MCAO) and cortical photothrombosis (PT) in
mice, we assessed post-stroke impairments via the Staircase
test. Lesion locations were identified using 7 T-MRI.
Machine learning was applied to reconstruct forepaw
kinematic trajectories and feature analysis was achieved
with MouseReach, a new data-processing toolbox. Lesion
reconstructions pinpointed ischemic centers in the striatum
(MCAO) and sensorimotor cortex (PT). Pellet retrieval
alterations were observed, but were unrelated to overall
stroke volume. Instead, forepaw slips and relative reaching
success correlated with increasing cortical lesion size in
both models. Striatal lesion size after MCAO was associated
with prolonged reach durations that occurred with delayed
symptom onset. Further analysis on the impact of selective
serotonin reuptake inhibitors in the PT model revealed no
clear treatment effects but replicated strong effect sizes
of slips for post-stroke deficit detection. In summary,
refined movement analysis unveiled specific deficits in two
widely-used mouse stroke models, emphasizing the value of
deep behavioral profiling in preclinical stroke research to
enhance model validity for clinical translation.},
keywords = {Animals / Mice / Disease Models, Animal / Male /
Infarction, Middle Cerebral Artery: diagnostic imaging /
Infarction, Middle Cerebral Artery: physiopathology /
Infarction, Middle Cerebral Artery: complications / Stroke:
physiopathology / Stroke: diagnostic imaging / Stroke:
complications / Mice, Inbred C57BL / Magnetic Resonance
Imaging: methods / Machine Learning / Movement: physiology /
Machine learning (Other) / motor deficits (Other) / rodent
models (Other) / stroke (Other) / translational research
(Other)},
cin = {AG Dirnagl / AG Endres},
ddc = {610},
cid = {I:(DE-2719)1810002 / I:(DE-2719)1811005},
pnm = {353 - Clinical and Health Care Research (POF4-353)},
pid = {G:(DE-HGF)POF4-353},
typ = {PUB:(DE-HGF)16},
pmc = {pmc:PMC11418716},
pubmed = {pmid:39234984},
doi = {10.1177/0271678X241254718},
url = {https://pub.dzne.de/record/271972},
}