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@ARTICLE{Pu:272883,
author = {Pu, Jingjing and Liu, Ting and Sharma, Amit and Jiang,
Liping and Wei, Feng and Ren, Xiubao and Schmidt-Wolf, Ingo
G H and Hou, Jian},
title = {{A}dvances in adoptive cellular immunotherapy and
therapeutic breakthroughs in multiple myeloma.},
journal = {Experimental hematology $\&$ oncology},
volume = {13},
number = {1},
issn = {2162-3619},
address = {London},
publisher = {Biomed Central},
reportid = {DZNE-2024-01299},
pages = {105},
year = {2024},
abstract = {The basic idea of modulating the immune system to better
recognize and fight tumor cells has led to the successful
introduction of adoptive cellular immunotherapy (ACT).
ACT-based treatment regimens, in which the patient's own
immune cells are isolated and subsequently expanded (ex
vivo) and reinfused, have also contributed significantly to
the development of a personalized treatment strategy.
Complementing this, the unprecedented advances in ACTs as
chimeric antigen receptor (CAR)-T cell therapies and their
derivatives such as CAR-NK, CAR-macrophages, CAR-γδT and
CAR-NKT have further maximized the therapeutic outcomes.
Herein, we provide a comprehensive overview of the
development of ACTs in multiple myeloma (MM) and outline how
they have evolved from an experimental form to a mainstay of
standard clinical settings. Besides, we provide insights
into cytokine-induced killer cell (CIK) therapy, an
alternative form of ACT that (as CIK or CAR-CIK) has
enormous potential in the clinical spectrum of MM. We also
summarize the results of the major preclinical and clinical
studies of adoptive cell therapy in MM and address the
current challenges (such as cytokine release syndrome (CRS)
and neurotoxicity) that limit its complete success in the
cancer landscape.},
subtyp = {Review Article},
keywords = {CAR-NK (Other) / CAR-T (Other) / CIK (Other) / Cell therapy
(Other) / Immunotherapy (Other) / Multiple myeloma (Other)},
cin = {AG Ehninger},
ddc = {610},
cid = {I:(DE-2719)1013005},
pnm = {352 - Disease Mechanisms (POF4-352)},
pid = {G:(DE-HGF)POF4-352},
typ = {PUB:(DE-HGF)16},
pmc = {pmc:PMC11514856},
pubmed = {pmid:39468695},
doi = {10.1186/s40164-024-00576-6},
url = {https://pub.dzne.de/record/272883},
}