% IMPORTANT: The following is UTF-8 encoded. This means that in the presence
% of non-ASCII characters, it will not work with BibTeX 0.99 or older.
% Instead, you should use an up-to-date BibTeX implementation like “bibtex8” or
% “biber”.
@ARTICLE{Lischewski:275887,
author = {Lischewski, Stella Andrea and Konrad, Kerstin and Dogan,
Imis and Didszun, Claire and Costa, Ana Sofia and Schawohl,
Sara Annabelle and Giunti, Paola and Parkinson, Michael H
and Mariotti, Caterina and Nanetti, Lorenzo and Durr,
Alexandra and Ewenczyk, Claire and Boesch, Sylvia and
Nachbauer, Wolfgang and Klopstock, Thomas and Stendel,
Claudia and de Rivera Garrido, Francisco Javier Rodríguez
and Schöls, Ludger and Fleszar, Zofia and Klockgether,
Thomas and Grobe-Einsler, Marcus and Giordano, Ilaria and
Rai, Myriam and Pandolfo, Massimo and Schulz, Jörg B and
Reetz, Kathrin},
collaboration = {group, EFACTS study},
othercontributors = {Indelicato, Elisabetta and Ampros, Matthias and Gellera,
Cinzia and Mongelli, Alessia and Castaldo, Anna and Fichera,
Mario and Bertini, Enrico and Vasco, Gessica and Biet, Marie
and Monin, Marie Lorraine and Holtbernd, Florian and Brcina,
Nikolina and Hohenfeld, Christian and Radelfahr, Florentine
and Bischoff, Almut T and Hayer, Stefanie N and Koutsis,
Georgios and Breza, Marianthi and Palau, Francesc and
O'Callaghan, Mar and Thomas-Black, Gilbert and Manso,
Katarina and Solanky, Nita and Labrum, Robyn},
title = {{L}ongitudinal analysis of anthropometric measures over 5
years in patients with {F}riedreich ataxia in the {EFACTS}
natural history study.},
journal = {European journal of neurology},
volume = {32},
number = {1},
issn = {1351-5101},
address = {Oxford [u.a.]},
publisher = {Wiley-Blackwell},
reportid = {DZNE-2025-00122},
pages = {e70011},
year = {2025},
abstract = {Friedreich ataxia is a rare neurodegenerative disorder
caused by frataxin deficiency. Both underweight and
overweight occur in mitochondrial disorders, each with
adverse health outcomes. We investigated the longitudinal
evolution of anthropometric abnormalities in Friedreich
ataxia and the hypothesis that both weight loss and weight
gain are associated with faster disease
progression.Participants were drawn from the European
Friedreich's Ataxia Consortium for Translational Studies
(EFACTS). Age- and sex-specific BMI and height scores were
calculated using the KIGGS-BMI percentiles for children.
Height correction was applied for scoliosis. Longitudinal
data were analysed using linear mixed effects models and
incremental standard deviation scores and growth mixture
models identified subclasses with varying BMI
trajectories.Five hundred and forty-three adults and
fifty-nine children were assessed for up to 5 years. In
children, severe underweight $(26\%),$ underweight $(7\%),$
severe short stature $(16\%)$ and short stature $(23\%)$
were common. The corrected BMI percentile was stable in
children, although $48\%$ had negative incremental BMI
scores over 1 year and $63\%$ over 3 years versus
$10\%/year$ in a normal reference cohort. Overweight was
common in adults $(19\%),$ with a slight increase in BMI
over time. Longer GAA repeat size was linked to lower BMI in
adults. Weight trajectory was not associated with ataxia
progression in adults.Significant anthropometric
abnormalities were identified, with underweight and short
stature prevalent in children and overweight in adults.
These findings highlight the need for regular nutritional
monitoring and interventions to manage underweight in
children and promote healthy weight in adults.},
keywords = {Humans / Friedreich Ataxia: epidemiology / Friedreich
Ataxia: complications / Friedreich Ataxia: physiopathology /
Male / Female / Longitudinal Studies / Adult / Child /
Adolescent / Disease Progression / Body Mass Index / Young
Adult / Thinness: epidemiology / Middle Aged / Anthropometry
/ Overweight: epidemiology / Overweight: complications /
Friedreich ataxia (Other) / body height (Other) / body mass
index (Other) / natural history (Other) / underweight
(Other)},
cin = {Clinical Research (Munich) / AG Schöls / AG Gasser /
Patient Studies (Bonn) / Clinical Research (Bonn)},
ddc = {610},
cid = {I:(DE-2719)1111015 / I:(DE-2719)5000005 /
I:(DE-2719)1210000 / I:(DE-2719)1011101 /
I:(DE-2719)1011001},
pnm = {353 - Clinical and Health Care Research (POF4-353)},
pid = {G:(DE-HGF)POF4-353},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:39797559},
pmc = {pmc:PMC11724196},
doi = {10.1111/ene.70011},
url = {https://pub.dzne.de/record/275887},
}