% IMPORTANT: The following is UTF-8 encoded. This means that in the presence
% of non-ASCII characters, it will not work with BibTeX 0.99 or older.
% Instead, you should use an up-to-date BibTeX implementation like “bibtex8” or
% “biber”.
@ARTICLE{Schirge:277548,
author = {Schirge, Philine Marie and Perneczky, Robert and Taoka,
Toshiaki and Ruiz-Rizzo, Adriana L and Ersoezlue, Ersin and
Forbrig, Robert and Guersel, Selim and Kurz, Carolin and
Brendel, Matthias and Hellmann-Regen, Julian and Priller,
Josef and Schneider, Anja and Jessen, Frank and Düzel,
Emrah and Buerger, Katharina and Teipel, Stefan and Laske,
Christoph and Peters, Oliver and Spruth, Eike and
Fliessbach, Klaus and Rostamzadeh, Ayda and Glanz, Wenzel
and Janowitz, Daniel and Kilimann, Ingo and Sodenkamp,
Sebastian and Ewers, Michael and Rauchmann, Boris-Stephan},
title = {{P}erivascular space and white matter hyperintensities in
{A}lzheimer's disease: associations with disease progression
and cognitive function.},
journal = {Alzheimer's research $\&$ therapy},
volume = {17},
number = {1},
issn = {1758-9193},
address = {London},
publisher = {BioMed Central},
reportid = {DZNE-2025-00437},
pages = {62},
year = {2025},
abstract = {Alzheimer's disease (AD) is the leading cause of dementia,
characterized by the accumulation of amyloid-beta (Aβ) and
neurofibrillary tangles. Recent studies emphasize the role
of vascular factors, including the glymphatic system, in AD
pathogenesis, particularly in Aβ clearance. The diffusion
tensor image analysis along the perivascular space
(DTI-ALPS; ALPS-Index) has emerged as a novel, non-invasive
method to evaluate the glymphatic system in vivo, showing
glymphatic insufficiency in AD. This study aimed to
investigate alterations in the function of the glymphatic
system in individuals with AD versus healthy controls (HC),
and to explore its association with Aβ, cerebrovascular
disease (CVD), white matter hyperintensities (WMH), and
cognitive function.DTI MRI data from three independent study
cohorts (ActiGliA: AD n = 16, Controls n = 18; DELCODE: AD n
= 54, Controls n = 67; ADNI: AD n = 43, Controls n = 49)
were used to evaluate the perivascular space (PVS)
integrity; a potential biomarker for glymphatic activity.
The DTI-Along the Perivascular Space technique was used to
measure water diffusion along PVS providing an index to
assess the efficiency of the glymphatic system's waste
clearance function. WMH load was quantified in FLAIR MRI
using the lesion segmentation tool. We quantified WMHs
volume within our defined region of interest (ROI) and
excluded participants with any WMHs to avoid confounding the
ALPS-Index. Associations with cerebrospinal fluid (CSF) AD
hallmark biomarkers, cognitive performance (MMSE) and
clinical severity (CDR) were assessed.AD patients had a
significantly lower ALPS-Index vs. healthy controls
(ActiGliA: AD: mean = 1.22, SD = 0.12; Controls: mean =
1.36, SD = 0.14, p = 0.004; DELCODE: AD: mean = 1.26, SD =
0.18; Controls: mean = 1.34, SD = 0.2, p = 0.035; ADNI: AD:
mean = 1.08, SD = 0.24; Controls: mean = 1.19, SD = 0.13, p
= 0.008). The ALPS-Index was associated with CSF Aβ
concentration, WMH number and MMSE and CDR. WMH, found in
the ROIs correlated negatively with the ALPS-Index.This
study highlights the potential of the DTI-ALPS-Index as a
biomarker for glymphatic dysfunction in AD. It underscores
the importance of considering vascular factors and the
glymphatic system in the pathogenesis and diagnosis of AD as
WMHs in the ROI could cause disturbances and inaccurate
indices.},
keywords = {Humans / Alzheimer Disease: diagnostic imaging / Alzheimer
Disease: pathology / Female / Male / White Matter:
diagnostic imaging / White Matter: pathology / Aged /
Disease Progression / Glymphatic System: diagnostic imaging
/ Glymphatic System: pathology / Diffusion Tensor Imaging:
methods / Cognition: physiology / Aged, 80 and over /
Amyloid beta-Peptides: cerebrospinal fluid / Amyloid
beta-Peptides: metabolism / Magnetic Resonance Imaging:
methods / Brain: diagnostic imaging / Brain: pathology /
Alzheimer's disease (Other) / Amyloid-beta (Other) /
Cognitive decline (Other) / Dementia (Other) / Diffusion
tensor imaging (Other) / Perivascular space (Other) /
Amyloid beta-Peptides (NLM Chemicals)},
cin = {AG Simons / AG Dichgans / Clinical Research (Munich) / AG
Dirnagl / AG Endres / AG Priller / AG Schneider / AG Jessen
/ Patient Studies (Bonn) / AG Düzel / AG Teipel / AG Gasser
/ ICRU},
ddc = {610},
cid = {I:(DE-2719)1110008 / I:(DE-2719)5000022 /
I:(DE-2719)1111015 / I:(DE-2719)1810002 / I:(DE-2719)1811005
/ I:(DE-2719)5000007 / I:(DE-2719)1011305 /
I:(DE-2719)1011102 / I:(DE-2719)1011101 / I:(DE-2719)5000006
/ I:(DE-2719)1510100 / I:(DE-2719)1210000 /
I:(DE-2719)1240005},
pnm = {351 - Brain Function (POF4-351) / 353 - Clinical and Health
Care Research (POF4-353) / 899 - ohne Topic (POF4-899)},
pid = {G:(DE-HGF)POF4-351 / G:(DE-HGF)POF4-353 /
G:(DE-HGF)POF4-899},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:40098158},
doi = {10.1186/s13195-025-01707-9},
url = {https://pub.dzne.de/record/277548},
}