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@ARTICLE{Talevi:278552,
author = {Talevi, Valentina and Lee, Hang-mao and Liu, Dan and Beyer,
Marc D and Salomoni, Paolo and Breteler, Monique M B and
Aziz, N Ahmad},
title = {{A}ge and {S}ex {E}ffects on {B}lood {R}etrotransposable
{E}lement {E}xpression {L}evels: {F}indings {F}rom the
{P}opulation-{B}ased {R}hineland {S}tudy.},
journal = {Aging cell},
volume = {24},
number = {8},
issn = {1474-9718},
address = {Oxford [u.a.]},
publisher = {Wiley-Blackwell},
reportid = {DZNE-2025-00592},
pages = {e70092},
year = {2025},
abstract = {Retrotransposable elements (RTEs) have been implicated in
the pathogenesis of several age-associated diseases.
Although model systems indicate that age- and sex-dependent
loss of heterochromatin increases RTE expression, data from
large human studies are lacking. Here we assessed the
expression levels of 795 blood RTE subfamilies in 2467
participants of the population-based Rhineland Study. We
found that the expression of more than $98\%$ of RTE
subfamilies increased with both chronological and biological
age. Moreover, the expression of heterochromatin regulators
involved in RTE silencing was negatively related to the
expression of 690 RTE subfamilies. Finally, we observed sex
differences in 42 RTE subfamilies, with higher expression in
men. The genes mapped to sex-related RTEs were enriched in
immune response-related pathways. Importantly, we validated
our key findings in an independent population-based cohort.
Our findings indicate that RTEs and their repressors are
markers of aging and that their dysregulation is linked to
inflammation, especially in men.},
keywords = {aging (Other) / biomarkers (Other) / heterochromatin
(Other) / immune response (Other) / population‐based study
(Other) / retrotransposable elements (Other) /
sex‐differences (Other)},
cin = {AG Aziz / AG Breteler / AG Liu / AG Beyer / AG Salomoni /
PRECISE},
ddc = {610},
cid = {I:(DE-2719)5000071 / I:(DE-2719)1012001 /
I:(DE-2719)1012009 / I:(DE-2719)1013035 / I:(DE-2719)1013032
/ I:(DE-2719)1013031},
pnm = {354 - Disease Prevention and Healthy Aging (POF4-354) / 351
- Brain Function (POF4-351) / 352 - Disease Mechanisms
(POF4-352) / TRANSIT-ND - Tandem Repeats Associated with
Neurogenomic Somatic Instability and Neurodegeneration
(101041677)},
pid = {G:(DE-HGF)POF4-354 / G:(DE-HGF)POF4-351 /
G:(DE-HGF)POF4-352 / G:(EU-Grant)101041677},
experiment = {EXP:(DE-2719)Rhineland Study-20190321 /
EXP:(DE-2719)PRECISE-20190321},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:40320676},
doi = {10.1111/acel.70092},
url = {https://pub.dzne.de/record/278552},
}