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@ARTICLE{Xu:279124,
author = {Xu, Jishu and Hörner, Michaela and Nagel, Maike and
Perhat, Perwin and Korneck, Milena and Noß, Marvin and
Hauser, Stefan and Schöls, Ludger and Admard, Jakob and
Nicolas, Casadei and Schüle-Freyer, Rebecca},
title = {{U}nravelling axonal transcriptional landscapes: insights
from induced pluripotent stem cell-derived cortical neurons
and implications for motor neuron degeneration},
journal = {Open biology},
volume = {15},
number = {6},
issn = {2046-2441},
address = {London},
publisher = {Royal Society Publishing},
reportid = {DZNE-2025-00684},
pages = {250101},
year = {2025},
abstract = {Neuronal function and pathology are deeply influenced by
the distinct molecular profiles of the axon and soma.
Traditional studies have often overlooked these differences
due to the technical challenges of compartment-specific
analysis. In this study, we employ a robust RNA-sequencing
approach, using microfluidic devices, to generate
high-quality axonal transcriptomes from induced pluripotent
stem cells-derived cortical neurons (CNs). We achieve high
specificity of axonal fractions, ensuring sample purity
without contamination. Comparative analysis revealed a
unique and specific transcriptional landscape in axonal
compartments, characterized by diverse transcript types,
including protein-coding mRNAs, RNAs encoding ribosomal
proteins, mitochondrial-encoded RNAs and long non-coding
RNAs. Previous works have reported the existence of
transcription factors (TFs) in the axon. Here, we detect a
set of TFs specific to the axon and indicative of their
active participation in transcriptional regulation. To
investigate transcripts and pathways essential for central
motor neuron (MN) degeneration and maintenance we analysed
kinesin family member 1C (KIF1C)-knockout (KO) CNs,
modelling hereditary spastic paraplegia, a disorder
associated with prominent length-dependent degeneration of
central MN axons. We found that several key factors crucial
for survival and health were absent in KIF1C-KO axons,
highlighting a possible role of these also in other
neurodegenerative diseases. Taken together, this study
underscores the utility of microfluidic devices in studying
compartment-specific transcriptomics in human neuronal
models and reveals complex molecular dynamics of axonal
biology. The impact of KIF1C on the axonal transcriptome not
only deepens our understanding of MN diseases but also
presents a promising avenue for exploration of
compartment-specific disease mechanisms.},
keywords = {Induced Pluripotent Stem Cells: metabolism / Induced
Pluripotent Stem Cells: cytology / Axons: metabolism / Motor
Neurons: metabolism / Motor Neurons: pathology / Animals /
Mice / Transcriptome / Humans / Gene Expression Profiling /
Transcription Factors: metabolism / Kinesins: genetics /
Kinesins: metabolism / Nerve Degeneration: genetics / Nerve
Degeneration: metabolism / axonal transcriptomics (Other) /
axonal transport (Other) / iPSC-derived neurons (Other) /
kinesin (Other) / neurons (Other) / transcription factors
(Other) / Transcription Factors (NLM Chemicals) / Kinesins
(NLM Chemicals)},
cin = {AG Hauser / AG Schöls},
ddc = {570},
cid = {I:(DE-2719)1210016 / I:(DE-2719)5000005},
pnm = {352 - Disease Mechanisms (POF4-352) / 353 - Clinical and
Health Care Research (POF4-353)},
pid = {G:(DE-HGF)POF4-352 / G:(DE-HGF)POF4-353},
typ = {PUB:(DE-HGF)16},
doi = {10.1098/rsob.250101},
url = {https://pub.dzne.de/record/279124},
}